Taylor Ping
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Researcher
Unknown Researcher
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Taylor Ping studies the evolutionary and ecological drivers of animal behavior and physiology, with a particular focus on predator-prey interactions and their impact on fitness and population dynamics. Their research investigates how predation pressure shapes adaptive plasticity in prey defense behaviors and digestive physiology, examining trade-offs between antipredator strategies and other life-history components such as growth and reproduction. Ping's work also explores the role of local adaptation in maintaining species coexistence within ecosystems, considering how selective pressures influence phenotypic traits. Collaborations include researchers from the University of Arkansas at Fayetteville, such as Adam M. Siepielski, with whom Ping has co-authored five publications. Ping's scholarship metrics include an h-index of 3, with 6 total publications and 60 citations.
Metrics
- h-index: 3
- Publications: 6
- Citations: 64
Selected Publications
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Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence (2026)
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Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence (2026)
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Cheating death: selection on digestive physiology overcomes expected growth costs of antipredator defences (2025)
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Predators drive selection for adaptive plasticity in prey defense behavior (2025)
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Interactions between fitness components across the life cycle constrain competitor coexistence (2023)
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A common measure of prey immune function is not constrained by the cascading effects of predators (2021)
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Predators weaken prey intraspecific competition through phenotypic selection (2020)
Collaboration Network
Top Collaborators
- Interactions between fitness components across the life cycle constrain competitor coexistence
- A common measure of prey immune function is not constrained by the cascading effects of predators
- Predators drive selection for adaptive plasticity in prey defense behavior
- Cheating death: selection on digestive physiology overcomes expected growth costs of antipredator defences
- Interactions between fitness components across the life cycle constrain competitor coexistence
- Predators drive selection for adaptive plasticity in prey defense behavior
- Cheating death: selection on digestive physiology overcomes expected growth costs of antipredator defences
- Interactions between fitness components across the life cycle constrain competitor coexistence
- A common measure of prey immune function is not constrained by the cascading effects of predators
- Predators drive selection for adaptive plasticity in prey defense behavior
- Cheating death: selection on digestive physiology overcomes expected growth costs of antipredator defences
- A common measure of prey immune function is not constrained by the cascading effects of predators
- Interactions between fitness components across the life cycle constrain competitor coexistence
- Cheating death: selection on digestive physiology overcomes expected growth costs of antipredator defences
- Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence
- Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence
- Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence
- Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence
- Evidence consistent with local adaptation to predation shaping stabilizing effects underlying local coexistence
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