JG Moore
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Also affiliated: BC Cancer Agency (1985); University College Hospital (2021); National Institutes of Health (1984–1987); Scripps Health (1985); United States Food and Drug Administration (2025); Johns Hopkins University (1985–1986); Stratasys (Israel) (1985–1986); Johns Hopkins Medicine (1986); Johns Hopkins Bayview Medical Center (1986); University Medical Center (1986); Public Health England (1984); Sustainable Home Survey (United Kingdom) (1984); National Heart, Lung, and Blood Institute (1984–1986); University Hospital and Clinics (1986); St. Mark's Hospital (2021); National Heart Hospital (1986); National Public Health Laboratory (1984); Frederick National Laboratory for Cancer Research (1985); National Institute of Allergy and Infectious Diseases (1985); Scripps Clinic (1985); University College London (2021); Imperial College London (2021)
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Biography and Research Information
OverviewAI-generated summary
JG Moore's research focuses on toxicological research, with a specific emphasis on the effects of substances on biological systems. Their work has explored the alteration of cytokine and chemokine transcript levels in the placenta of mouse models treated prenatally with dexamethasone. Moore has also investigated phenotypic diversity in relation to disease models, specifically a novel bowel disease model utilizing organoids. Their scholarship includes 13 publications with 903 citations, and an h-index of 8. Moore has collaborated with Noriko Nakamura on shared publications, indicating a network within the National Center for Toxicological Research.
Metrics
- h-index: 8
- Publications: 13
- Citations: 904
Selected Publications
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Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice (2026)
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Alteration of Cytokine/Chemokine Transcript Levels in the Placenta of Humanized Mouse Models Treated Prenatally With Dexamethasone (2025)
Collaboration Network
Top Collaborators
- Alteration of Cytokine/Chemokine Transcript Levels in the Placenta of Humanized Mouse Models Treated Prenatally With Dexamethasone
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
- Alteration of Cytokine/Chemokine Transcript Levels in the Placenta of Humanized Mouse Models Treated Prenatally With Dexamethasone
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
- Alteration of Cytokine/Chemokine Transcript Levels in the Placenta of Humanized Mouse Models Treated Prenatally With Dexamethasone
- Alteration of Cytokine/Chemokine Transcript Levels in the Placenta of Humanized Mouse Models Treated Prenatally With Dexamethasone
- Alteration of Cytokine/Chemokine Transcript Levels in the Placenta of Humanized Mouse Models Treated Prenatally With Dexamethasone
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
- Potential Effects of Prepubertal Exposure to Perfluorooctane Sulfonic Acid on the First Wave of Folliculogenesis in Young CD ‐1 Mice
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