Match tier Likely match
Presence Current · Arkansas
Last published 2020
Sources OpenAlex · ORCID
Refreshed 2026-10-10

Michael A. Cozart

This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.

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8 h-index 15 pubs 281 cited

  • Animals
  • Male
  • Mice
  • Female
  • Mice, Transgenic
  • Leptin
  • Receptors, Leptin
  • Disease Models, Animal
  • Mice, Inbred C57BL
  • Humans
  • Time Factors
  • Body Composition
  • Insulin
  • Obesity
  • Binding Sites

Biography and Research Information

Metrics

  • h-index: 8
  • Publications: 15
  • Citations: 281

Selected Publications

  • Vascular smooth muscle TRPC3 channels facilitate the inverse hemodynamic response during status epilepticus (2020)
    Scientific Reports 17 citations DOI OpenAlex
  • Elucidating the Involvement of Endothelial TRPC3 Channels in Neurovascular Coupling During Status Epilepticus (2018)
    The FASEB Journal DOI OpenAlex
  • TRPC 3 channels play a critical role in the theta component of pilocarpine‐induced status epilepticus in mice (2016)
    Epilepsia 37 citations DOI OpenAlex
  • Hippocampal administration of chondroitinase ABC increases plaque-adjacent synaptic marker and diminishes amyloid burden in aged APPswe/PS1dE9 mice (2015)
    Acta Neuropathologica Communications 56 citations DOI OpenAlex
  • A retrospective review of the progress in amyotrophic lateral sclerosis drug discovery over the last decade and a look at the latest strategies (2015)
    Expert Opinion on Drug Discovery 31 citations DOI OpenAlex
  • Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility (2014)
    Endocrinology 44 citations DOI OpenAlex
  • Ghrelin Restoration of Function In Vitro in Somatotropes from Male Mice Lacking the Janus Kinase (JAK)-Binding Site of the Leptin Receptor (2013)
    Endocrinology 16 citations DOI OpenAlex
  • Functional defects in mice somatotropes caused by ablation of leptin receptors is reversed, in vitro, by stimulation with Ghrelin (2012)
    The FASEB Journal 1 citation DOI OpenAlex
  • Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity (2012)
    Endocrinology 23 citations DOI OpenAlex
  • The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity (2010)
    Endocrinology 53 citations DOI OpenAlex

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