Andrea A. Duina Data-verified
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Biography and Research Information
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Andrea A. Duina's research focuses on the molecular mechanisms of gene regulation and chromatin dynamics, primarily utilizing the model organism *Saccharomyces cerevisiae* (budding yeast). Her work investigates the interplay between histone modifications, transcriptional machinery, and DNA sequences in controlling gene expression. Recent publications examine the impact of specific histone mutations, such as H3-L61R, on the interactions of the transcription factor Spt16 with genes and explore how DNA sequences at the 3' end of genes influence the occupancy of transcription factors like yFACT and RNA polymerase II, as well as nucleosome positioning.
Duina has received federal funding for her work, including an NSF grant totaling $83,387 for a conference aimed at broadening research opportunities within South Central EPSCoR states. She is recognized as a federal grant principal investigator. Her research collaborations include work with Elijah G. Dilday, Alex Pablo-Kaiser, McKenzie G. Tucker, and Avery G. Olmstead, all from Hendrix College, with whom she has co-authored publications. Duina maintains an active laboratory website to disseminate her research findings and laboratory activities. Her scholarly contributions are reflected in an h-index of 13, with 29 total publications and 993 citations.
Metrics
- h-index: 13
- Publications: 29
- Citations: 996
Selected Publications
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Trapping of yFACT at 3’ ends of genes is not a universal characteristic of yeast versions of Bryant-Li-Bhoj syndrome histone H3 mutants (2024)
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Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy (2024)
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Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast (2022)
Federal Grants 1 $83,387 total
Conference: Broadening Access to Research Opportunities in South Central EPSCoR States
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- Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast
- Trapping of yFACT at 3’ ends of genes is not a universal characteristic of yeast versions of Bryant-Li-Bhoj syndrome histone H3 mutants
- Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast
- Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast
- Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast
- Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast
- Dominant effects of the histone mutant H3-L61R on Spt16-gene interactions in budding yeast
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
- Assessing contributions of DNA sequences at the 3’ end of a yeast gene on yFACT, RNA polymerase II, and nucleosome occupancy
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