Biography and Research Information
OverviewAI-generated summary
Charlie Wright's research focuses on medication management and its impact on patient care, particularly within the context of neurological disorders and aging populations. Wright has published on the importance of medication reconciliation for patients with Parkinson's disease, highlighting potential challenges and best practices. Their work also extends to fundamental molecular mechanisms, with publications addressing dysregulated signaling pathways in osteosarcoma and the analysis of gene expression programs in neuroblastoma. These diverse publications indicate an interest in both clinical applications of pharmacology and the underlying biological processes of disease.
Metrics
- Publications: 3
Selected Publications
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The Importance of Medication Reconciliation in a Patient with Parkinson’s Disease (2025)
Collaboration Network
Top Collaborators
- Abstract 4075: Meta-analysis of neuroblastoma single cell RNA-seq datasets identifies conserved and divergent gene expression programs across human and preclinical models
- Abstract 4075: Meta-analysis of neuroblastoma single cell RNA-seq datasets identifies conserved and divergent gene expression programs across human and preclinical models
- Abstract 4075: Meta-analysis of neuroblastoma single cell RNA-seq datasets identifies conserved and divergent gene expression programs across human and preclinical models
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
- Abstract B043: Dysregulated AKT signaling reprograms osteosarcoma to drive selective reliance on EP300
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