Howard H. Conaway
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Also affiliated: United States Department of Veterans Affairs (1974); University of Arkansas Medical Center (1974); Central Arkansas Veterans Healthcare System (1981); Harry S. Truman Memorial Veterans' Hospital (1974); John L. McClellan Memorial Veterans Hospital (1981); University of Missouri (1972–1974)
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Howard H. Conaway's research has focused on the study of metabolic and genetic disorders. His work has investigated the underlying mechanisms of these conditions, aiming to understand their progression and potential therapeutic targets. Conaway has contributed to the scientific literature through publications detailing his findings in this area.
Metrics
- h-index: 6
- Publications: 9
- Citations: 181
Selected Publications
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Activation of Shc1 Allows Oncostatin M to Induce RANKL and Osteoclast Formation More Effectively Than Leukemia Inhibitory Factor (2019)
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Plasma lipids in a colony of spontaneously diabetic New Zealand White rabbits (1981)
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Spontaneous diabetes mellitus in the New Zealand white rabbit: Physiologic characteristics (1981)
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Depression of Leucine and Isoproterenol Induced Insulin Secretions in the Spontaneously Diabetic New Zealand White Rabbit (1980)
Collaboration Network
Top Collaborators
- Spontaneous diabetes mellitus in the New Zealand white rabbit: Physiologic characteristics
- Plasma lipids in a colony of spontaneously diabetic New Zealand White rabbits
- Spontaneous diabetes mellitus in the New Zealand white rabbit: Physiologic characteristics
- Spontaneous diabetes mellitus in the New Zealand white rabbit: Physiologic characteristics
- Depression of Leucine and Isoproterenol Induced Insulin Secretions in the Spontaneously Diabetic New Zealand White Rabbit
- Depression of Leucine and Isoproterenol Induced Insulin Secretions in the Spontaneously Diabetic New Zealand White Rabbit
- Plasma lipids in a colony of spontaneously diabetic New Zealand White rabbits
- Activation of Shc1 Allows Oncostatin M to Induce RANKL and Osteoclast Formation More Effectively Than Leukemia Inhibitory Factor
- Activation of Shc1 Allows Oncostatin M to Induce RANKL and Osteoclast Formation More Effectively Than Leukemia Inhibitory Factor
- Activation of Shc1 Allows Oncostatin M to Induce RANKL and Osteoclast Formation More Effectively Than Leukemia Inhibitory Factor
- Activation of Shc1 Allows Oncostatin M to Induce RANKL and Osteoclast Formation More Effectively Than Leukemia Inhibitory Factor
- Activation of Shc1 Allows Oncostatin M to Induce RANKL and Osteoclast Formation More Effectively Than Leukemia Inhibitory Factor
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