Mohammad Goodarzi Data-verified
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Biography and Research Information
OverviewAI-generated summary
Mohammad Goodarzi's research focuses on the application of quantitative structure-activity relationship (QSAR) models and advanced analytical techniques to understand molecular interactions and biological processes. His work has involved the development and calibration of algorithms for analyzing complex datasets, including those derived from near-infrared spectroscopy. He has explored molecular modeling and discriminant analysis to predict outcomes in various biological systems, utilizing both animal and human data.
His publications span diverse areas, including the molecular mechanisms of gene regulation, the metabolomics of infectious diseases, and advances in organic reaction methodologies. He has also contributed to case reviews in medical conditions and investigated novel anticoagulation methods. His research network includes collaborations on studies of vascular smooth muscle function and protein-protein interaction networks in microorganisms.
Goodarzi holds a strong publication record with an h-index of 28 and over 2,800 citations. He is recognized as a highly cited researcher, indicating a significant impact in his fields of study. He maintains an active laboratory website to disseminate his research activities.
Metrics
- h-index: 28
- Publications: 127
- Citations: 2,841
Selected Publications
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Unlocking the Potential of Bacteriophage Endolysins: A Promising Alternative for Combating Antibiotic Resistance in Poultry (2025)
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Metabolomics of volatile organic compounds (VOCs) in infectious diseases (2024)
Collaboration Network
Top Collaborators
- <i>Wnt16</i>Promotes Vascular Smooth Muscle Contractile Phenotype and Function via Taz (Wwtr1) Activation in Male<i>LDLR−/−</i>Mice
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Abstract 562: Altered Hippo Pathway Activity And Impaired Smooth Muscle Contractile Phenotype In Wnt16-Deficient Mice Worsens Ascending Aortic Aneurysm
- <i>Wnt16</i>Promotes Vascular Smooth Muscle Contractile Phenotype and Function via Taz (Wwtr1) Activation in Male<i>LDLR−/−</i>Mice
- Abstract 562: Altered Hippo Pathway Activity And Impaired Smooth Muscle Contractile Phenotype In Wnt16-Deficient Mice Worsens Ascending Aortic Aneurysm
- <i>Wnt16</i>Promotes Vascular Smooth Muscle Contractile Phenotype and Function via Taz (Wwtr1) Activation in Male<i>LDLR−/−</i>Mice
- Abstract 562: Altered Hippo Pathway Activity And Impaired Smooth Muscle Contractile Phenotype In Wnt16-Deficient Mice Worsens Ascending Aortic Aneurysm
- <i>Wnt16</i>Promotes Vascular Smooth Muscle Contractile Phenotype and Function via Taz (Wwtr1) Activation in Male<i>LDLR−/−</i>Mice
- Abstract 562: Altered Hippo Pathway Activity And Impaired Smooth Muscle Contractile Phenotype In Wnt16-Deficient Mice Worsens Ascending Aortic Aneurysm
- <i>Wnt16</i>Promotes Vascular Smooth Muscle Contractile Phenotype and Function via Taz (Wwtr1) Activation in Male<i>LDLR−/−</i>Mice
- Abstract 562: Altered Hippo Pathway Activity And Impaired Smooth Muscle Contractile Phenotype In Wnt16-Deficient Mice Worsens Ascending Aortic Aneurysm
- <i>Wnt16</i>Promotes Vascular Smooth Muscle Contractile Phenotype and Function via Taz (Wwtr1) Activation in Male<i>LDLR−/−</i>Mice
- Abstract 562: Altered Hippo Pathway Activity And Impaired Smooth Muscle Contractile Phenotype In Wnt16-Deficient Mice Worsens Ascending Aortic Aneurysm
- Global protein-protein interaction networks in yeast saccharomyces cerevisiae and helicobacter pylori
- Corrigendum to ’Global protein-protein interaction networks in yeast saccharomyces cerevisiae and helicobacter pylori’ [Talanta 265 (2023) 124836]
- Global protein-protein interaction networks in yeast saccharomyces cerevisiae and helicobacter pylori
- Corrigendum to ’Global protein-protein interaction networks in yeast saccharomyces cerevisiae and helicobacter pylori’ [Talanta 265 (2023) 124836]
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
- Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration
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