Perla Rocha-Loyola Data-verified
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Researcher
unknown
Research Areas
Biography and Research Information
OverviewAI-generated summary
Perla Rocha-Loyola's research focuses on the interactions between plants and bacterial pathogens, specifically investigating how bacterial proteins influence plant cellular processes and immune responses. Her work has examined the role of Pseudomonas syringae type III effectors, such as HopG1 and HopD1, in triggering or interfering with plant cell death and immune functions. She has explored the involvement of plant proteins, like AtNHR2B, in modulating these pathogen-induced responses. Rocha-Loyola's scholarship includes three publications, with her most recent work in 2023 detailing the interference of the effector HopD1 with cellular dynamics related to the plant immune protein AtNHR2B. She has maintained recent activity in her field, evidenced by her 2022 and 2023 publications. Her scholarly network includes collaborators from the University of Arkansas at Fayetteville, such as Clemencia M. Rojas and Catalina Rodríguez-Puerto, with whom she has co-authored multiple publications.
Metrics
- h-index: 2
- Publications: 3
- Citations: 21
Selected Publications
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The Pseudomonas syringae pv. tomato DC3000 effector HopD1 interferes with cellular dynamics associated with the function of the plant immune protein AtNHR2B (2023)
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The Pseudomonas syringae type III effector HopG1 triggers necrotic cell death that is attenuated by AtNHR2B (2022)
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<i>Pseudomonas syringae</i> pv. tomato DC3000 Effector HopG1 is a multi-faceted protein that Triggers Necrotic Cell Death that is attenuated by the Nonhost Resistance 2B (AtNHR2B) Protein (2021)
Collaboration Network
Top Collaborators
- The Pseudomonas syringae type III effector HopG1 triggers necrotic cell death that is attenuated by AtNHR2B
- The Pseudomonas syringae pv. tomato DC3000 effector HopD1 interferes with cellular dynamics associated with the function of the plant immune protein AtNHR2B
- <i>Pseudomonas syringae</i> pv. tomato DC3000 Effector HopG1 is a multi-faceted protein that Triggers Necrotic Cell Death that is attenuated by the Nonhost Resistance 2B (AtNHR2B) Protein
- The Pseudomonas syringae type III effector HopG1 triggers necrotic cell death that is attenuated by AtNHR2B
- The Pseudomonas syringae pv. tomato DC3000 effector HopD1 interferes with cellular dynamics associated with the function of the plant immune protein AtNHR2B
- <i>Pseudomonas syringae</i> pv. tomato DC3000 Effector HopG1 is a multi-faceted protein that Triggers Necrotic Cell Death that is attenuated by the Nonhost Resistance 2B (AtNHR2B) Protein
- The Pseudomonas syringae type III effector HopG1 triggers necrotic cell death that is attenuated by AtNHR2B
- <i>Pseudomonas syringae</i> pv. tomato DC3000 Effector HopG1 is a multi-faceted protein that Triggers Necrotic Cell Death that is attenuated by the Nonhost Resistance 2B (AtNHR2B) Protein
- The Pseudomonas syringae type III effector HopG1 triggers necrotic cell death that is attenuated by AtNHR2B
- <i>Pseudomonas syringae</i> pv. tomato DC3000 Effector HopG1 is a multi-faceted protein that Triggers Necrotic Cell Death that is attenuated by the Nonhost Resistance 2B (AtNHR2B) Protein
- The Pseudomonas syringae pv. tomato DC3000 effector HopD1 interferes with cellular dynamics associated with the function of the plant immune protein AtNHR2B
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