M. W. Shaw

Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.

High Impact

Professor of Plant Disease Ecology

University of Arkansas

Also affiliated: University of East Anglia (1982–1987); Centers for Disease Control and Prevention (1998–2000); New Mexico Department of Health (2000); Google (United States) (1995); Met Office (1969); National Board of Medical Examiners (2000); Michigan Department of Natural Resources (2025); University of Arizona (1993); Natural Environment Research Council (2003); National Institute of Agricultural Botany (2003); The University of Melbourne (2004); University of New Mexico (2000); Universidad de Granada (1997); Vanderbilt University (1984); Agricultural Development Advisory Service (United Kingdom) (2003–2013); D. E. Shaw Research (1998–2003); Rothamsted Research (2003–2013); Arizona Western College (1993); The Royal Free Hospital (2025); University of Bristol (1985–2009); UK Centre for Ecology & Hydrology (2003); North Yorkshire County Council (2003); National Institute for Public Health and the Environment (2004–2013); National Institute of Public Health (2004); Cambridge Quantum Computing (United Kingdom) (2008–2013); Institute for Advanced Study (1982); Anderson Hospital (1973); Roland Hill (United Kingdom) (2025); The University of Texas Health Science Center (1982); National Center for Infectious Diseases (1998–2000); University College London (2025); University of York (1983–1986); University of Reading (1989–2025); Michigan State University (2025); Harper Adams University (1946); The University of Texas Health Science Center at Houston (2008)

Faculty Researcher

44 h-index 228 pubs 6,863 cited

Research Areas

Biomedical Subjects

Links

ORCID Lab Website Research Group

OverviewAI-generated summary

M. W. Shaw's research program focuses on plant disease ecology, with an emphasis on biological and chemical control strategies. Shaw has investigated the use of microbial agents, such as *Bacillus subtilis* and *Aureobasidium pullulans*, for managing post-harvest rot in cherries caused by *Monilinia laxa*. This work includes field applications and studies on suppressing overwintering inoculum on mummified fruits. Additionally, Shaw's research has explored the spatial spread of plant pathogens like *Ditylenchus gigas* and their interactions with other microbes on crops such as *Vicia faba*.

Further research extends to the mechanisms of fungicide resistance in plant pathogens. Shaw has modeled how dose splitting can influence the selection for both target-site and non-target-site fungicide resistance, a critical consideration for sustainable agriculture. The researcher has also examined symptomless growth patterns of fungi, such as *Botrytis cinerea*, in host plants like *Lactuca sativa* and *Arabidopsis thaliana*. Shaw's work is supported by a significant publication record, with 227 total publications and an h-index of 44, indicating a substantial contribution to the field. Key collaborators at the University of Arkansas at Fayetteville include Lauren L. Berry and Caleb P. Roberts.