Stacey D. Rodenbeck
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Assistant Professor of Biology
Also affiliated: Indiana University School of Medicine (2016–2021); Indiana University – Purdue University Indianapolis (2016–2021)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Stacey D. Rodenbeck's research investigates the mechanisms of coronary artery disease, focusing on intracellular calcium dysregulation in coronary smooth muscle. Her work utilizes animal models, specifically Ossabaw miniature swine, to study disease progression and identify similarities with human coronary artery disease. Rodenbeck's publications examine multiphasic changes in smooth muscle calcium transporters during the development of atherosclerosis. She has authored 12 publications with approximately 99 citations and an h-index of 4. Rodenbeck is an Assistant Professor of Biology at Harding University.
Metrics
- h-index: 4
- Publications: 12
- Citations: 102
Selected Publications
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Multiphasic changes in smooth muscle Ca2+ transporters during the progression of coronary atherosclerosis (2022)
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Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine (2021)
Collaboration Network
Top Collaborators
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Multiphasic changes in smooth muscle Ca2+ transporters during the progression of coronary atherosclerosis
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Multiphasic changes in smooth muscle Ca2+ transporters during the progression of coronary atherosclerosis
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Intracellular Ca2+ Dysregulation in Coronary Smooth Muscle Is Similar in Coronary Disease of Humans and Ossabaw Miniature Swine
- Multiphasic changes in smooth muscle Ca2+ transporters during the progression of coronary atherosclerosis
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