Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Nora E. Gibbons' research focuses on the genetics and viability of bacteria, with a specific emphasis on *Borrelia burgdorferi*, the causative agent of Lyme disease. Her work investigates essential genes and gene expression regulation within this bacterium. Gibbons has explored the role of specific gene families, such as paralogous gene family 52, in the enzootic cycle of *Borrelia burgdorferi*, finding that plasmid-encoded members of this family are dispensable for this process. Her research also touches upon DNA-binding proteins and their function in microbial systems. Gibbons has collaborated with researchers including Jon S. Blevins, Brendan P. Moore, and William Bellamy at the University of Arkansas for Medical Sciences. Her scholarship metrics include an h-index of 1, with 2 total publications and 57 total citations.
Metrics
- h-index: 1
- Publications: 2
- Citations: 57
Selected Publications
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The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i> (2024)
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Rrp2, a Prokaryotic Enhancer-Like Binding Protein, Is Essential for Viability of Borrelia burgdorferi (2012)
Collaboration Network
Top Collaborators
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
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