William Bellamy
Researcher
Also affiliated: City Of Hope National Medical Center (2005); University of Arizona (1988–2006); Johns Hopkins University (2020); Prudential Financial (United States) (2018); University of South Florida (2004); University of Arkansas Medical Center (2017); Arizona Science Center (1992); Moffitt Cancer Center (2004–2006); Public Health England (2007–2014); University of Illinois Chicago (2005); Biology of Infection (2007); City of Hope (2005); Health Protection Agency (2014); Palmetto Hematology Oncology (2004); Banner - University Medical Center Tucson (1996); Ventana Research Corporation (United States) (2000); University of Virginia Medical Center (1983); Arizona Cancer Therapeutics (United States) (1988–1989); Arizona Oncology (1988–2001); University of Arizona Cancer Center (2000); University of Virginia (1982); University of Cincinnati (2000)
Faculty Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
William Bellamy is a researcher at the University of Arkansas for Medical Sciences with a focus on disease mechanisms and microbial infections. His work has led to 93 total publications, which have been cited approximately 4,756 times, contributing to an h-index of 30. Bellamy is recognized as a highly cited researcher. His recent publication in 2024 investigates the role of plasmid-encoded genes in the enzootic cycle of *Borrelia burgdorferi*. He has collaborated with colleagues including Jon S. Blevins, Brendan P. Moore, and Nora E. Gibbons, all from the University of Arkansas for Medical Sciences, on shared publications.
Metrics
- h-index: 30
- Publications: 93
- Citations: 4,762
Selected Publications
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The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i> (2024)
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Analysis of primary central nervous system large B-cell lymphoma in the era of high-grade B-cell lymphoma: Detection of two cases with MYC and BCL6 rearrangements in a cohort of 12 cases (2020)
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ALK Rearrangements Are Infrequent in Cellular Blue Nevus and Deep Penetrating Nevus (2018)
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Short-Duration Rapid Chilling of Mastectomy Specimens Does Not Interfere With Breast Cancer Biomarker and Molecular Testing (2018)
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The level of deletion 17p and bi-allelic inactivation of <i>TP53</i> has a significant impact on clinical outcome in multiple myeloma (2017)
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BB0238, a Presumed Tetratricopeptide Repeat-Containing Protein, Is Required during Borrelia burgdorferi Mammalian Infection (2014)
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Chitosan coating to enhance the therapeutic efficacy of calcium sulfate-based antibiotic therapy in the treatment of chronic osteomyelitis (2014)
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Ex Vivo–expanded Natural Killer Cells Demonstrate Robust Proliferation In Vivo in High-risk Relapsed Multiple Myeloma Patients (2014)
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Highly activated and expanded natural killer cells for multiple myeloma immunotherapy (2012)
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Use of Xylitol To Enhance the Therapeutic Efficacy of Polymethylmethacrylate-Based Antibiotic Therapy in Treatment of Chronic Osteomyelitis (2012)
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Evaluation of Dynamic [18F]-FDG-PET Imaging for the Detection of Acute Post-Surgical Bone Infection (2012)
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Autologous Expanded Natural Killer Cells As a New Therapeutic Option for High-Risk Myeloma (2011)
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Phase II randomized trial of bevacizumab <i>versus</i> bevacizumab and thalidomide for relapsed/refractory multiple myeloma: a California Cancer Consortium trial (2011)
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A phase II trial of single agent bevacizumab in patients with relapsed, aggressive non-Hodgkin lymphoma: Southwest oncology group study S0108 (2009)
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Expression of Myeloma-Specific Markers in Bone Marrow Spicules Using a Novel Immunohistochemical Technique (2008)
Collaboration Network
Top Collaborators
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
- The plasmid-encoded members of paralogous gene family 52 are dispensable to the enzootic cycle of <i>Borrelia burgdorferi</i>
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