Sankalpa Chakraborty
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Ph.D. Candidacy Proposal Defense
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Sankalpa Chakraborty's research investigates molecular mechanisms underlying cancer and cellular processes. His work has explored the role of cytoglobin in tumor hypoxia and its connection to cancer suppression, as well as how DNA damage can activate cytoglobin to stabilize p53 and induce G1 cell cycle arrest. Chakraborty has also examined the biology of FOXM1 and its potential in cancer therapy. More recently, his research has focused on transcriptomic interactions of noncoding RNAs in the heart under altered thyroid hormonal states and the noncoding and coding mechanisms of aging heart failure with preserved ejection fraction in the context of thyroid dysfunction. He has also contributed to studies on intracellular trafficking and glycosylation in plant cells and the identification of protein interactors in yeast.
Metrics
- h-index: 2
- Publications: 11
- Citations: 12
Selected Publications
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Abstract 4368964: LncRNA Regulated by Triiodothyronine Confers Cardioprotection in High-Fat-Diet-Induced Atherosclerosis-Driven Myocardial Infarction (2025)
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Noncoding and coding mechanisms of aging-related heart failure with preserved ejection fraction associated with thyroid dysfunction (2025)
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Prenylated Stilbenoids Confer Sex-Dependent Cardioprotection in Rats (2025)
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Noncoding and Coding Mechanisms of Aging Heart Failure with Preserved Ejection Fraction with Thyroid Dysfunction (2025)
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Abstract Mo115: Aging Heart Failure with Preserved Ejection Fraction is Mediated by Noncoding RNAs (2024)
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Intracellular trafficking and glycosylation of hydroxyproline-O-glycosylation module in tobacco BY-2 cells is dependent on medium composition and transcriptome analysis (2023)
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Novel Interactomes of Cardiac Noncoding RNA Transcriptome under Altered Thyroid Hormonal States (2023)
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Novel Transcriptomic Interactomes of Noncoding RNAs in the Heart under Altered Thyroid Hormonal States (2023)
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Long Non‐coding RNAs Mediate Heart Failure with Preserved Ejection Fraction Associated with Thyroid Dysfunction (2022)
Collaboration Network
Top Collaborators
- Novel Transcriptomic Interactomes of Noncoding RNAs in the Heart under Altered Thyroid Hormonal States
- Noncoding and Coding Mechanisms of Aging Heart Failure with Preserved Ejection Fraction with Thyroid Dysfunction
- Long Non‐coding RNAs Mediate Heart Failure with Preserved Ejection Fraction Associated with Thyroid Dysfunction
- Novel Interactomes of Cardiac Noncoding RNA Transcriptome under Altered Thyroid Hormonal States
- Abstract Mo115: Aging Heart Failure with Preserved Ejection Fraction is Mediated by Noncoding RNAs
Showing 5 of 8 shared publications
- Noncoding and Coding Mechanisms of Aging Heart Failure with Preserved Ejection Fraction with Thyroid Dysfunction
- Long Non‐coding RNAs Mediate Heart Failure with Preserved Ejection Fraction Associated with Thyroid Dysfunction
- Abstract Mo115: Aging Heart Failure with Preserved Ejection Fraction is Mediated by Noncoding RNAs
- Noncoding and coding mechanisms of aging-related heart failure with preserved ejection fraction associated with thyroid dysfunction
- Noncoding and Coding Mechanisms of Aging Heart Failure with Preserved Ejection Fraction with Thyroid Dysfunction
- Long Non‐coding RNAs Mediate Heart Failure with Preserved Ejection Fraction Associated with Thyroid Dysfunction
- Abstract Mo115: Aging Heart Failure with Preserved Ejection Fraction is Mediated by Noncoding RNAs
- Noncoding and coding mechanisms of aging-related heart failure with preserved ejection fraction associated with thyroid dysfunction
- Noncoding and Coding Mechanisms of Aging Heart Failure with Preserved Ejection Fraction with Thyroid Dysfunction
- Abstract Mo115: Aging Heart Failure with Preserved Ejection Fraction is Mediated by Noncoding RNAs
- Noncoding and coding mechanisms of aging-related heart failure with preserved ejection fraction associated with thyroid dysfunction
- Noncoding and Coding Mechanisms of Aging Heart Failure with Preserved Ejection Fraction with Thyroid Dysfunction
- Abstract Mo115: Aging Heart Failure with Preserved Ejection Fraction is Mediated by Noncoding RNAs
- Noncoding and coding mechanisms of aging-related heart failure with preserved ejection fraction associated with thyroid dysfunction
- Novel Transcriptomic Interactomes of Noncoding RNAs in the Heart under Altered Thyroid Hormonal States
- Novel Interactomes of Cardiac Noncoding RNA Transcriptome under Altered Thyroid Hormonal States
- Noncoding and coding mechanisms of aging-related heart failure with preserved ejection fraction associated with thyroid dysfunction
- Abstract 4368964: LncRNA Regulated by Triiodothyronine Confers Cardioprotection in High-Fat-Diet-Induced Atherosclerosis-Driven Myocardial Infarction
- Long Non‐coding RNAs Mediate Heart Failure with Preserved Ejection Fraction Associated with Thyroid Dysfunction
- Intracellular trafficking and glycosylation of hydroxyproline-O-glycosylation module in tobacco BY-2 cells is dependent on medium composition and transcriptome analysis
- Intracellular trafficking and glycosylation of hydroxyproline-O-glycosylation module in tobacco BY-2 cells is dependent on medium composition and transcriptome analysis
- Intracellular trafficking and glycosylation of hydroxyproline-O-glycosylation module in tobacco BY-2 cells is dependent on medium composition and transcriptome analysis
- Intracellular trafficking and glycosylation of hydroxyproline-O-glycosylation module in tobacco BY-2 cells is dependent on medium composition and transcriptome analysis
- Intracellular trafficking and glycosylation of hydroxyproline-O-glycosylation module in tobacco BY-2 cells is dependent on medium composition and transcriptome analysis
- Prenylated Stilbenoids Confer Sex-Dependent Cardioprotection in Rats
- Prenylated Stilbenoids Confer Sex-Dependent Cardioprotection in Rats
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