Andrea Powers
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Also affiliated: Ottawa Public Health (2021); Indiana University School of Medicine (2007–2013); Indiana University – Purdue University Indianapolis (2007–2013)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Andrea Powers' research has focused on the molecular mechanisms underlying organ development, particularly the lacrimal gland. Her work investigates the role of fibroblast growth factor (FGF) signaling pathways, including FGF10 and its receptor FGFR2b, in embryonic induction and tissue formation. Powers has explored how heparan sulfate modifications, specifically N-sulfation and O-sulfation, regulate FGF diffusion and signaling efficiency. Additionally, her research has examined the involvement of intracellular signaling molecules like Shp2 and Kras in these developmental processes. Powers has also published on topics related to gene regulation and cellular function, including the role of lanthionine synthase C-like protein-1 in redox regulation and retinoid exposure's effect on gene expression in cutaneous T-cell lymphoma.
Metrics
- h-index: 6
- Publications: 9
- Citations: 338
Selected Publications
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77283 All-Trans Retinoic Acid Downregulates FOXP3 Expression in Cutaneous T-Cell Lymphoma (2026)
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Stable knockout of lanthionine synthase C-like protein-1 (LanCL1) from HeLa cells indicates a role for LanCL1 in redox regulation of deubiquitinating enzymes (2020)
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Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma (2020)
Collaboration Network
Top Collaborators
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- 77283 All-Trans Retinoic Acid Downregulates FOXP3 Expression in Cutaneous T-Cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- 77283 All-Trans Retinoic Acid Downregulates FOXP3 Expression in Cutaneous T-Cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- 77283 All-Trans Retinoic Acid Downregulates FOXP3 Expression in Cutaneous T-Cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Retinoid Exposure Decreases Foxp3 Expression in Cutaneous T‐cell Lymphoma
- Stable knockout of lanthionine synthase C-like protein-1 (LanCL1) from HeLa cells indicates a role for LanCL1 in redox regulation of deubiquitinating enzymes
- Stable knockout of lanthionine synthase C-like protein-1 (LanCL1) from HeLa cells indicates a role for LanCL1 in redox regulation of deubiquitinating enzymes
- Stable knockout of lanthionine synthase C-like protein-1 (LanCL1) from HeLa cells indicates a role for LanCL1 in redox regulation of deubiquitinating enzymes
- Stable knockout of lanthionine synthase C-like protein-1 (LanCL1) from HeLa cells indicates a role for LanCL1 in redox regulation of deubiquitinating enzymes
- Stable knockout of lanthionine synthase C-like protein-1 (LanCL1) from HeLa cells indicates a role for LanCL1 in redox regulation of deubiquitinating enzymes
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