H. Herschel Conaway
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Also affiliated: Baylor College of Medicine (1982); University of Arkansas Medical Center (1975); UConn Health (1986); University of Missouri (1971–1973); Umeå University (2002–2006)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
H. Herschel Conaway's research focuses on the mechanisms of bone resorption and osteoclast formation. His work investigates the influence of various hormones and cellular factors on these processes. Recent publications include studies on the synergistic interactions between glucocorticoids, parathyroid hormone, and vitamin D3 in stimulating osteoclast activity, as well as the role of Vitamin A in enhancing periosteal osteoclastogenesis and its association with tissue-derived macrophages and osteoclast progenitors.
Conaway's scholarship metrics include an h-index of 17, with a total of 37 publications and 1,665 citations. He is actively publishing, with his most recent work appearing in 2024.
Metrics
- h-index: 17
- Publications: 36
- Citations: 1,655
Selected Publications
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Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors (2024)
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Stimulation of osteoclast formation and bone resorption by glucocorticoids: Synergistic interactions with the calcium regulating hormones parathyroid hormone and 1,25(OH)2-vitamin D3 (2022)
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Glucocorticoids employ the monomeric glucocorticoid receptor to potentiate vitamin D 3 and parathyroid hormone‐induced osteoclastogenesis (2019)
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Clinically relevant doses of vitamin A decrease cortical bone mass in mice (2018)
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Effects of retinoids on physiologic and inflammatory osteoclastogenesis in vitro (2018)
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Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity (2016)
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Retinoid Receptors in Bone and Their Role in Bone Remodeling (2015)
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Vitamin A Metabolism, Action, and Role in Skeletal Homeostasis (2013)
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Inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A (2012)
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Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor (2011)
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Stimulation of mouse calvarial periosteal resorption and inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A (2011)
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Interleukin-4 and interleukin-13 inhibit the expression of leukemia inhibitory factor and interleukin-11 in fibroblasts (2011)
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Retinoids inhibit differentiation of hematopoetic osteoclast progenitors (2009)
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Glucocorticoid Regulation of Osteoclast Differentiation and Expression of Receptor Activator of Nuclear Factor-κB (NF-κB) Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvarial Bones (2006)
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Stimulation of Resorption in Cultured Mouse Calvarial Bones by Thiazolidinediones (2005)
Collaboration Network
Top Collaborators
- IL-6, Leukemia Inhibitory Factor, and Oncostatin M Stimulate Bone Resorption and Regulate the Expression of Receptor Activator of NF-κB Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvariae
- Vitamin A Metabolism, Action, and Role in Skeletal Homeostasis
- Inhibition of Hormone and Cytokine-stimulated Osteoclastogenesis and Bone Resorption by Interleukin-4 and Interleukin-13 Is Associated with Increased Osteoprotegerin and Decreased RANKL and RANK in a STAT6-dependent Pathway
- Glucocorticoid Regulation of Osteoclast Differentiation and Expression of Receptor Activator of Nuclear Factor-κB (NF-κB) Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvarial Bones
- Retinoid Receptors in Bone and Their Role in Bone Remodeling
Showing 5 of 20 shared publications
- Vitamin A Metabolism, Action, and Role in Skeletal Homeostasis
- Retinoid Receptors in Bone and Their Role in Bone Remodeling
- Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor
- Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity
- Clinically relevant doses of vitamin A decrease cortical bone mass in mice
Showing 5 of 11 shared publications
- IL-6, Leukemia Inhibitory Factor, and Oncostatin M Stimulate Bone Resorption and Regulate the Expression of Receptor Activator of NF-κB Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvariae
- Inhibition of Hormone and Cytokine-stimulated Osteoclastogenesis and Bone Resorption by Interleukin-4 and Interleukin-13 Is Associated with Increased Osteoprotegerin and Decreased RANKL and RANK in a STAT6-dependent Pathway
- Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor
- Retinoids inhibit differentiation of hematopoetic osteoclast progenitors
- Stimulation of Resorption in Cultured Mouse Calvarial Bones by Thiazolidinediones
- Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor
- Retinoids inhibit differentiation of hematopoetic osteoclast progenitors
- Stimulation of mouse calvarial periosteal resorption and inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A
- Inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A
- Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor
- Retinoids inhibit differentiation of hematopoetic osteoclast progenitors
- Stimulation of mouse calvarial periosteal resorption and inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A
- IL-6, Leukemia Inhibitory Factor, and Oncostatin M Stimulate Bone Resorption and Regulate the Expression of Receptor Activator of NF-κB Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvariae
- Inhibition of Hormone and Cytokine-stimulated Osteoclastogenesis and Bone Resorption by Interleukin-4 and Interleukin-13 Is Associated with Increased Osteoprotegerin and Decreased RANKL and RANK in a STAT6-dependent Pathway
- Interleukin-4 and interleukin-13 inhibit the expression of leukemia inhibitory factor and interleukin-11 in fibroblasts
- Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor
- Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity
- Glucocorticoids employ the monomeric glucocorticoid receptor to potentiate vitamin D 3 and parathyroid hormone‐induced osteoclastogenesis
- Clinically relevant doses of vitamin A decrease cortical bone mass in mice
- Effects of retinoids on physiologic and inflammatory osteoclastogenesis in vitro
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Clinically relevant doses of vitamin A decrease cortical bone mass in mice
- Effects of retinoids on physiologic and inflammatory osteoclastogenesis in vitro
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Spontaneous diabetes mellitus in the New Zealand white rabbit
- Spontaneous Diabetes Mellitus in a Captive Golden-mantled Ground Squirrel, Spermophilus lateralis (Say)
- Spontaneous diabetes mellitus in the New Zealand white rabbit
- Hepatic ultrastructure in leprechaunism
- Retinoids inhibit differentiation of hematopoetic osteoclast progenitors
- Stimulation of Resorption in Cultured Mouse Calvarial Bones by Thiazolidinediones
- Enhancement of fetal rat limb bone resorption by phorbol ester (PMA) and ionophore A-23187
- Effects of forskolin on bone resorption in the absence and presence of parathyroid hormone and calcitonin
- Inhibition of Hormone and Cytokine-stimulated Osteoclastogenesis and Bone Resorption by Interleukin-4 and Interleukin-13 Is Associated with Increased Osteoprotegerin and Decreased RANKL and RANK in a STAT6-dependent Pathway
- Interleukin-4 and interleukin-13 inhibit the expression of leukemia inhibitory factor and interleukin-11 in fibroblasts
- Inhibition of Hormone and Cytokine-stimulated Osteoclastogenesis and Bone Resorption by Interleukin-4 and Interleukin-13 Is Associated with Increased Osteoprotegerin and Decreased RANKL and RANK in a STAT6-dependent Pathway
- Interleukin-4 and interleukin-13 inhibit the expression of leukemia inhibitory factor and interleukin-11 in fibroblasts
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