H. Herschel Conaway
Researcher
Also affiliated: University City Science Center (1987); Baylor College of Medicine (1982); University of Arkansas Medical Center (1975); UConn Health (1986); University of Missouri (1971–1973); University of Pennsylvania (1987); Umeå University (2002–2006)
Faculty Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
H. Herschel Conaway's research focuses on the mechanisms of bone resorption and osteoclast formation. His work investigates the influence of various hormones and cellular factors on these processes. Recent publications include studies on the synergistic interactions between glucocorticoids, parathyroid hormone, and vitamin D3 in stimulating osteoclast activity, as well as the role of Vitamin A in enhancing periosteal osteoclastogenesis and its association with tissue-derived macrophages and osteoclast progenitors.
Conaway's scholarship metrics include an h-index of 17, with a total of 37 publications and 1,665 citations. He is actively publishing, with his most recent work appearing in 2024.
Metrics
- h-index: 17
- Publications: 37
- Citations: 1,671
Selected Publications
-
Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors (2024)
-
Stimulation of osteoclast formation and bone resorption by glucocorticoids: Synergistic interactions with the calcium regulating hormones parathyroid hormone and 1,25(OH)2-vitamin D3 (2022)
-
Glucocorticoids employ the monomeric glucocorticoid receptor to potentiate vitamin D <sub>3</sub> and parathyroid hormone‐induced osteoclastogenesis (2019)
-
Clinically relevant doses of vitamin A decrease cortical bone mass in mice (2018)
-
Effects of retinoids on physiologic and inflammatory osteoclastogenesis in vitro (2018)
-
Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity (2016)
-
Retinoid Receptors in Bone and Their Role in Bone Remodeling (2015)
-
Vitamin A Metabolism, Action, and Role in Skeletal Homeostasis (2013)
-
Inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A (2012)
-
Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor (2011)
-
Stimulation of mouse calvarial periosteal resorption and inhibition of osteoclast formation in human peripheral CD14+ cells by vitamin A (2011)
-
Interleukin-4 and interleukin-13 inhibit the expression of leukemia inhibitory factor and interleukin-11 in fibroblasts (2011)
-
Retinoids inhibit differentiation of hematopoetic osteoclast progenitors (2009)
-
Glucocorticoid Regulation of Osteoclast Differentiation and Expression of Receptor Activator of Nuclear Factor-κB (NF-κB) Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvarial Bones (2006)
-
Stimulation of Resorption in Cultured Mouse Calvarial Bones by Thiazolidinediones (2005)
Collaboration Network
Top Collaborators
- Stimulation of osteoclast formation and bone resorption by glucocorticoids: Synergistic interactions with the calcium regulating hormones parathyroid hormone and 1,25(OH)2-vitamin D3
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Stimulation of osteoclast formation and bone resorption by glucocorticoids: Synergistic interactions with the calcium regulating hormones parathyroid hormone and 1,25(OH)2-vitamin D3
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
- Vitamin A enhanced periosteal osteoclastogenesis is associated with increased number of tissue-derived macrophages/osteoclast progenitors
Similar Researchers
Based on overlapping research topics