Cam Patterson
Chancellor
Also affiliated: Rutgers, The State University of New Jersey (2012); University of North Carolina at Chapel Hill (2000–2021); Texas Tech University (2003); Brigham and Women's Hospital (1995–2012); Novartis (Switzerland) (2007); Centre National de la Recherche Scientifique (2012); National Institutes of Health (2005); Mayo Clinic (2012); University of North Carolina Health Care (2009–2018); NewYork–Presbyterian Hospital (2014–2018); Howard Hughes Medical Institute (2012); University of Wisconsin–Madison (2012); Harvard University (1995–2012); North Carolina State University (2010); Johns Hopkins University (1996–2005); Max Planck Society (2013); Emory University (1997–2011); Université de Bordeaux (2012); University of Freiburg (2005–2014); Chungbuk National University (2017); Montclair State University (2012); Duke University (2004–2018); University of Alabama (2006); German Cancer Research Center (2008); University of California, San Francisco (2012); Baylor College of Medicine (2017); Regeneron (United States) (2007); Chinese Academy of Medical Sciences & Peking Union Medical College (2011); Vanderbilt University (2000); Cornell University (2017); Heidelberg University (1998–2002); Johns Hopkins Medicine (2005); University of Manchester (2012); University of Rochester Medicine (2010–2012); Boston Scientific (United States) (2006); University of Alabama at Birmingham (2000); University of North Carolina Hospitals (2014); National Cancer Institute (2002–2006); Institut de Biochimie et Génétique Cellulaires (2012); Mayo Clinic in Florida (2006–2012); Center for Cancer Research (2002); Harvard Stem Cell Institute (2012); Weill Cornell Medicine (2006–2017); UNC Lineberger Comprehensive Cancer Center (2000–2013); Novartis Institutes for BioMedical Research (2007); Weill Cornell Medical Center (2014–2018); University of Rochester (2010–2012); The University of Texas Medical Branch at Galveston (1997–2014); Rice University (1998–2001); McMaster University (1992)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Cam Patterson's research focuses on cellular mechanisms of protein degradation and stress response, particularly involving the ubiquitin-proteasome system and chaperone proteins. His work has investigated the role of the co-chaperone CHIP (CHIP) in regulating protein triage decisions mediated by heat-shock proteins, and how CHIP targets immature CFTR for proteasomal degradation. He has also studied the E3 ligase MuRF1 and its role in degrading myosin heavy chain protein in dexamethasone-treated skeletal muscle, as well as the high-affinity HSP90-CHIP complex's recognition and degradation of phosphorylated tau client proteins.
Patterson is recognized as a highly cited researcher, with a substantial publication record and over 43,000 citations. His scholarly output includes foundational guidelines for the use and interpretation of assays for monitoring autophagy. He has collaborated with several researchers at the University of Arkansas for Medical Sciences, including Kristie Hadden and Stephanie M. Gardner, with whom he shares multiple publications.
His research interests also extend to telomere length and its prediction of replicative capacity in human fibroblasts. Patterson maintains an active laboratory website, indicating ongoing research activities.
Metrics
- h-index: 94
- Publications: 595
- Citations: 42,822
Positions
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Chancellor publications 2018–2025University of Arkansas for Medical Sciences Institution web page
Selected Publications
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IL-28A/IL-10Rβ axis promotes angiogenesis via eNOS/AKT signaling and AP-1/NF-κB/MMP-2 network by regulating HSP70-1 expression (2024)
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Unique Responsibilities and Challenges of the Lone Academic Medical Center (2024)
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Design and Impact of a Novel Rural Hospital Alliance (2024)
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CRAT links cholesterol metabolism to innate immune responses in the heart (2023)
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Susan S. Smyth (1965–2022) (2023)
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RESPONSE: Last Fair Deal Gone Down (2022)
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From Strategic Planning to Strategy Impact (2021)
Collaboration Network
Top Collaborators
- From Strategic Planning to Strategy Impact
- Design and Impact of a Novel Rural Hospital Alliance
- Unique Responsibilities and Challenges of the Lone Academic Medical Center
- From Strategic Planning to Strategy Impact
- Design and Impact of a Novel Rural Hospital Alliance
- Unique Responsibilities and Challenges of the Lone Academic Medical Center
- Emerging Roles of Vascular Endothelium in Metabolic Homeostasis
- CRAT links cholesterol metabolism to innate immune responses in the heart
- Emerging Roles of Vascular Endothelium in Metabolic Homeostasis
- CRAT links cholesterol metabolism to innate immune responses in the heart
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
- Disrupted structure and aberrant function of CHIP mediates the loss of motor and cognitive function in preclinical models of SCAR16
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