C D Collins
Researcher
Also affiliated: Royal Marsden NHS Foundation Trust (1993–1995); Hammersmith Hospital (1995); St. Vincent's University Hospital (1997); Royal Marsden Hospital (1993–1995); Chelsea and Westminster Hospital (1992–1993)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
C D Collins' research focuses on the mechanisms of disease and medical imaging techniques. Current work includes investigating the correlation between non-alcoholic steatohepatitis and hepatocellular carcinoma in patients within a rural state. Another publication explores how the lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for Staphylococcus aureus. Collins' work has resulted in 15 publications and 285 citations, with an h-index of 5. Collaborations include shared publications with researchers at the University of Arkansas for Medical Sciences, including Stefanie Howell, Mark S. Smeltzer, and Karen E. Beenken, as well as Samantha Robinson at the University of Arkansas for Fayetteville.
Metrics
- h-index: 5
- Publications: 15
- Citations: 289
Selected Publications
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Menopausal status modifies waitlist outcomes in women with metabolic-associated steatotic liver disease under MELD 3.0 allocation (2026)
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Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i> (2026)
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Correlation between non-alcoholic Steatohepatitis and Hepatocellular carcinoma in patients in a Rural State (2025)
Collaboration Network
Top Collaborators
- Correlation between non-alcoholic Steatohepatitis and Hepatocellular carcinoma in patients in a Rural State
- Correlation between non-alcoholic Steatohepatitis and Hepatocellular carcinoma in patients in a Rural State
- Correlation between non-alcoholic Steatohepatitis and Hepatocellular carcinoma in patients in a Rural State
- Correlation between non-alcoholic Steatohepatitis and Hepatocellular carcinoma in patients in a Rural State
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Lipolysis of host triacylglyceride-rich lipoproteins creates a toxic microenvironment for <i>Staphylococcus aureus</i>
- Menopausal status modifies waitlist outcomes in women with metabolic-associated steatotic liver disease under MELD 3.0 allocation
- Menopausal status modifies waitlist outcomes in women with metabolic-associated steatotic liver disease under MELD 3.0 allocation
- Menopausal status modifies waitlist outcomes in women with metabolic-associated steatotic liver disease under MELD 3.0 allocation
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