Jordan Fett
Researcher
Also affiliated: Arkansas Children's Hospital (2021); Michigan United (2016); Michigan State University (2016)
Unknown Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Jordan Fett's research investigates the molecular and physiological underpinnings of genetic disorders affecting connective tissues and their impact on respiratory function. Fett's work has focused on mouse models to study diseases such as osteogenesis imperfecta and Ehlers-Danlos syndrome. These studies examine how alterations in type I collagen, a key structural protein, contribute to intrinsic lung and respiratory defects. Recent publications by Fett have detailed specific collagen gene haploinsufficiencies, such as in *Col5a1*, and their resultant phenotypes in these animal models. Fett also has experience with medical case reports, including a case of Eikenella Corrodens causing cystic fibrosis exacerbation and a neonate with cyanosis. Fett's scholarship metrics include an h-index of 3, with 5 total publications and 31 total citations. Key collaborators include Roy Morello and Milena Dimori from the University of Arkansas for Medical Sciences.
Metrics
- h-index: 3
- Publications: 5
- Citations: 31
Selected Publications
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Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta (2022)
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Haploinsufficiency of <i>Col5a1</i> causes intrinsic lung and respiratory changes in a mouse model of classical Ehlers‐Danlos syndrome (2022)
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A term neonate with cyanosis with crying (2021)
Collaboration Network
Top Collaborators
- Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta
- Haploinsufficiency of <i>Col5a1</i> causes intrinsic lung and respiratory changes in a mouse model of classical Ehlers‐Danlos syndrome
- Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta
- Haploinsufficiency of <i>Col5a1</i> causes intrinsic lung and respiratory changes in a mouse model of classical Ehlers‐Danlos syndrome
- Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta
- Haploinsufficiency of <i>Col5a1</i> causes intrinsic lung and respiratory changes in a mouse model of classical Ehlers‐Danlos syndrome
- A term neonate with cyanosis with crying
- A term neonate with cyanosis with crying
- A term neonate with cyanosis with crying
- A term neonate with cyanosis with crying
- A term neonate with cyanosis with crying
- Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta
- Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta
- Distinct type I collagen alterations cause intrinsic lung and respiratory defects of variable severity in mouse models of osteogenesis imperfecta
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