Josiah K. Leong Data-verified
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Biostatistician
faculty
Research Areas
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Biography and Research Information
OverviewAI-generated summary
Josiah K. Leong's research focuses on the application of biostatistical methods to neuroscience, particularly in areas related to brain structure, function, and behavior. His work has investigated the relationship between brain imaging data, such as magnetic resonance imaging, and various behavioral outcomes, including risk preference, externalizing problems, and relapse to stimulant drug use. Leong has contributed to studies examining specific brain regions like the nucleus accumbens and white matter, and their roles in human behavior across different age groups, from adolescence to adulthood.
He has also been involved in projects developing and utilizing computational platforms for neuroscience research, such as the brainlife.io cloud platform. His publications explore topics including the radiogenomics of genetic expansion carriers, the prediction of thalamic atrophy, and the analysis of socioaffective symptoms related to reward circuitry. Leong's scholarship metrics include an h-index of 11 with 34 publications and 470 citations. He has collaborated with researchers like Zach J. Gray and Jennifer C. Veilleux at the University of Arkansas at Fayetteville.
Metrics
- h-index: 11
- Publications: 35
- Citations: 489
Selected Publications
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Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults (2026)
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Structural projections to the nucleus accumbens link to impulsive components of human risk preference (2024)
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A Virtual In Vivo Dissection and Analysis of Socioaffective Symptoms Related to Cerebellum-Midbrain Reward Circuitry in Humans (2024)
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Author Correction: brainlife.io: a decentralized and open-source cloud platform to support neuroscience research (2024)
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Sex-Specific Vulnerability to Externalizing Problems: Sensitivity to Early Stress and Nucleus Accumbens Activation Over Adolescence (2024)
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brainlife.io: a decentralized and open-source cloud platform to support neuroscience research (2024)
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An <i>in vivo</i> Dissection, and Analysis of Socio-Affective Symptoms related to Cerebellum-Midbrain Reward Circuitry in Humans (2023)
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Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment (2022)
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Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment (2022)
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Brain tract structure predicts relapse to stimulant drug use (2022)
Collaboration Network
Top Collaborators
- Brain tract structure predicts relapse to stimulant drug use
- Structural projections to the nucleus accumbens link to impulsive components of human risk preference
- Structure of connections to the nucleus accumbens link to specific but not general measures of human risk preference
- Brain tract structure predicts relapse to stimulant drug use
- Structural projections to the nucleus accumbens link to impulsive components of human risk preference
- Structure of connections to the nucleus accumbens link to specific but not general measures of human risk preference
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Lifetime Stressor Exposure Profiles and Trait Risk for Substance Use in Young Adults
- Brain tract structure predicts relapse to stimulant drug use
- Structure of connections to the nucleus accumbens link to specific but not general measures of human risk preference
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
- Radiogenomics of<i>C9orf72</i>Expansion Carriers Reveals Global Transposable Element Derepression and Enables Prediction of Thalamic Atrophy and Clinical Impairment
- Radiogenomics of <i>C9orf72</i> expansion carriers reveals global transposable element de-repression and enables prediction of thalamic atrophy and clinical impairment
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