Biography and Research Information
OverviewAI-generated summary
Justin R. Nickell's research focuses on the neuropharmacological effects of substances, particularly in relation to vesicular monoamine transporter-2 (VMAT2) inhibition. His work investigates how VMAT2 inhibitors can mitigate the toxic effects of stimulants like methamphetamine on dopamine systems. A recent publication demonstrates that the VMAT2 inhibitor JPC-141 effectively prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rat models. Nickell's scholarship includes 33 publications with 639 citations and an h-index of 14. He has collaborated with researchers including Peter A. Crooks, Cassie M. Chandler, John P. Culver, and A. George Wilson at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 14
- Publications: 33
- Citations: 642
Selected Publications
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Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats (2024)
Collaboration Network
Top Collaborators
- Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats
- Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats
- Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats
- Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats
- Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats
- Vesicular monoamine transporter-2 inhibitor JPC-141 prevents methamphetamine-induced dopamine toxicity and blocks methamphetamine self-administration in rats
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