Match tier
Likely match
Presence
Current · Arkansas
Last published
2019
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
K
Kevin D. Young
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
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2 h-index
7 pubs
155 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Kevin D. Young's research has focused on microbial infections and disease research, with a particular emphasis on the development of antimicrobial resistance. His work has investigated the genetic mechanisms and biotechnological applications related to bacteria. Young has also contributed to studies on health sciences research and education, exploring the broader landscape of medical knowledge dissemination and application.
Metrics
- h-index: 2
- Publications: 7
- Citations: 155
Selected Publications
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Faculty Opinions recommendation of Regulation of the peptidoglycan polymerase activity of pbp1b by antagonist actions of the core divisome proteins ftsblq and ftsn. (2019)
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Faculty Opinions recommendation of Mycobacterial cell wall synthesis inhibitors cause lethal ATP burst. (2018)
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Faculty Opinions recommendation of Comprehensive analysis of central carbon metabolism illuminates connections between nutrient availability, growth rate, and cell morphology in Escherichia coli. (2018)
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A Defective Undecaprenyl Pyrophosphate Synthase Induces Growth and Morphological Defects That Are Suppressed by Mutations in the Isoprenoid Pathway of Escherichia coli (2018)
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Faculty Opinions recommendation of Crosstalk between the tricarboxylic acid cycle and peptidoglycan synthesis in Caulobacter crescentus through the homeostatic control of α-ketoglutarate. (2017)
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Dead‐end intermediates in the enterobacterial common antigen pathway induce morphological defects in Escherichia coli by competing for undecaprenyl phosphate (2015)
Collaboration Network
Top Collaborators
Matthew A. Jorgenson
University of Arkansas for Medical Sciences
5 shared publications
In database
- Dead‐end intermediates in the enterobacterial common antigen pathway induce morphological defects in Escherichia coli by competing for undecaprenyl phosphate
- Faculty Opinions recommendation of Regulation of the peptidoglycan polymerase activity of pbp1b by antagonist actions of the core divisome proteins ftsblq and ftsn.
- Faculty Opinions recommendation of Comprehensive analysis of central carbon metabolism illuminates connections between nutrient availability, growth rate, and cell morphology in Escherichia coli.
- Faculty Opinions recommendation of Mycobacterial cell wall synthesis inhibitors cause lethal ATP burst.
- Faculty Opinions recommendation of Crosstalk between the tricarboxylic acid cycle and peptidoglycan synthesis in Caulobacter crescentus through the homeostatic control of α-ketoglutarate.
Suresh Kannan
University of Arkansas for Medical Sciences (US)
2 shared publications
- Dead‐end intermediates in the enterobacterial common antigen pathway induce morphological defects in Escherichia coli by competing for undecaprenyl phosphate
- A Defective Undecaprenyl Pyrophosphate Synthase Induces Growth and Morphological Defects That Are Suppressed by Mutations in the Isoprenoid Pathway of Escherichia coli
Mary E. Laubacher
University of Arkansas for Medical Sciences (US)
1 shared publication
- Dead‐end intermediates in the enterobacterial common antigen pathway induce morphological defects in Escherichia coli by competing for undecaprenyl phosphate
William J. MacCain
University of Arkansas for Medical Sciences (US)
1 shared publication
- A Defective Undecaprenyl Pyrophosphate Synthase Induces Growth and Morphological Defects That Are Suppressed by Mutations in the Isoprenoid Pathway of Escherichia coli
Dannah Z. Jameel
University of North Carolina at Charlotte (US)
1 shared publication
- A Defective Undecaprenyl Pyrophosphate Synthase Induces Growth and Morphological Defects That Are Suppressed by Mutations in the Isoprenoid Pathway of Escherichia coli
Jerry M. Troutman
University of North Carolina at Charlotte (US)
1 shared publication
- A Defective Undecaprenyl Pyrophosphate Synthase Induces Growth and Morphological Defects That Are Suppressed by Mutations in the Isoprenoid Pathway of Escherichia coli
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