Mateen Ullah Data-verified
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Biography and Research Information
OverviewAI-generated summary
Mateen Ullah's research focuses on the development and characterization of various sensor materials, particularly for humidity detection. His work includes investigating the electrical and hysteretic properties of organic compound-based humidity sensors, as well as exploring the frequency and humidity-dependent sensing characteristics of sensors utilizing Schiff bases and Zinc(II) bis(dithiocarbamate) materials. Ullah has also published on the incipient ferroelectricity of AgTaO3 and the ferroelectric nature of (Li0.008Ag0.992)TaO3. His research interests extend to analyzing the impact of variables on infrastructure project participation using machine learning techniques. Ullah has a h-index of 6, with 27 total publications and 228 total citations.
Metrics
- h-index: 6
- Publications: 30
- Citations: 233
Selected Publications
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Genome-wide association study and genomic prediction of fruit weight in spinach (2026)
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Genome-Wide Characterization of VDAC Gene Family in Soybean (Glycine max L.) and In Silico Expression Profiling in Response to Drought and Salt Stress (2025)
Collaboration Network
Top Collaborators
- Electrical and hysteric properties of organic compound-based humidity sensor and its dualistic interactive approach to H2O molecules
- Frequency and humidity dependent sensing properties of 3-(4, 5-diphenyl-1H-imidazol-2-yl)phenol based humidity sensor
- Low hysteresis, high sensitivity, fast response, and recovery time of humidity sensor based on Schiff bases material
- Synthesis and characterization of Zinc(II) bis(dithiocarbamate) humidity sensors
- Enhanced dielectric and electrical properties of Li-doped BNBT ceramics for energy storage applications
- Incipient ferroelectricity of AgTaO3 and ferroelectric nature of (Li0.008Ag0.992)TaO3
- Enhanced dielectric and electrical properties of Li-doped BNBT ceramics for energy storage applications
- Dielectric, strain engineering and microstructural studies of La modified (Bi0.5Na0.5)0.94 Ba0.06TiO3 relaxor ferroelectrics
- Investigation of structural, electrical and strain properties of [(Bi0.5Na0.5)0.935Ba0.065]1-Zn TiO3 ceramics
- Electrical and hysteric properties of organic compound-based humidity sensor and its dualistic interactive approach to H2O molecules
- Frequency and humidity dependent sensing properties of 3-(4, 5-diphenyl-1H-imidazol-2-yl)phenol based humidity sensor
- Low hysteresis, high sensitivity, fast response, and recovery time of humidity sensor based on Schiff bases material
- Synthesis and characterization of Zinc(II) bis(dithiocarbamate) humidity sensors
- Electrical and hysteric properties of organic compound-based humidity sensor and its dualistic interactive approach to H2O molecules
- Frequency and humidity dependent sensing properties of 3-(4, 5-diphenyl-1H-imidazol-2-yl)phenol based humidity sensor
- Low hysteresis, high sensitivity, fast response, and recovery time of humidity sensor based on Schiff bases material
- Synthesis and characterization of Zinc(II) bis(dithiocarbamate) humidity sensors
- Frequency and humidity dependent sensing properties of 3-(4, 5-diphenyl-1H-imidazol-2-yl)phenol based humidity sensor
- Low hysteresis, high sensitivity, fast response, and recovery time of humidity sensor based on Schiff bases material
- Synthesis and characterization of Zinc(II) bis(dithiocarbamate) humidity sensors
- Enhanced dielectric and electrical properties of Li-doped BNBT ceramics for energy storage applications
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Supplementary Material for: A novel intronic deletion in PDE6B causes autosomal recessive retinitis pigmentosa by interfering with RNA splicing
- Enhanced dielectric and electrical properties of Li-doped BNBT ceramics for energy storage applications
- Dielectric, strain engineering and microstructural studies of La modified (Bi0.5Na0.5)0.94 Ba0.06TiO3 relaxor ferroelectrics
- Investigation of structural, electrical and strain properties of [(Bi0.5Na0.5)0.935Ba0.065]1-Zn TiO3 ceramics
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