Charles K. Lumpkin
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Also affiliated: Arkansas Children's Hospital (1991–2016); Amgen (United States) (2002–2005); United States Department of Veterans Affairs (1986); State University of New York (1994); Baylor College of Medicine (1985–1986); Virginia Commonwealth University (1982–1983); Veterans Health Administration (1986); W. Alton Jones Cell Science Center (1980); University at Albany, State University of New York (1994); Central Arkansas Veterans Healthcare System (1985); National Institute on Aging (1991); Arkansas Children's Nutrition Center (2006–2013); John L. McClellan Memorial Veterans Hospital (1983–1994); Arkansas Children's Research Institute (1995–2016); California State University, Long Beach (2012); Virginia Commonwealth University Medical Center (1982–1983)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Charles K. Lumpkin's research has focused on the mechanisms of viral pathogenesis and host immune responses. His work has investigated how viruses interact with cellular machinery to promote their replication and spread, as well as the body's defense mechanisms against viral infections. He has published studies detailing the molecular interactions between viruses and host cells. His research has contributed to understanding the complex interplay between pathogens and the immune system.
Metrics
- h-index: 37
- Publications: 76
- Citations: 4,068
Selected Publications
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Corrigendum to “SGLT2 inhibitor therapy improves blood glucose but does not prevent diabetic bone disease in diabetic DBA/2J male mice” [Bone 82 (2016) 101–107] (2016)
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The impact of SGLT2 inhibitors, compared with insulin, on diabetic bone disease in a mouse model of type 1 diabetes (2016)
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Nutlin‐3 treatment spares cisplatin‐induced inhibition of bone healing while maintaining osteosarcoma toxicity (2016)
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SGLT2 inhibitor therapy improves blood glucose but does not prevent diabetic bone disease in diabetic DBA/2J male mice (2015)
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Effects of long-term doxycycline on bone quality and strength in diabetic male DBA/2J mice (2015)
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Loss of Insulin Receptor in Osteoprogenitor Cells Impairs Structural Strength of Bone (2014)
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Loss of Functional NADPH Oxidase 2 Protects Against Alcohol‐Induced Bone Resorption in Female p47phox −/− Mice (2013)
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Osteo-promoting effects of insulin-like growth factor I (IGF-I) in a mouse model of type 1 diabetes (2013)
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Cisplatin inhibits bone healing during distraction osteogenesis (2013)
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Vitamin D Supplementation Protects against Bone Loss Associated with Chronic Alcohol Administration in Female Mice (2012)
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Dysregulation of the Intrarenal Vitamin D Endocytic Pathway in a Nephropathy-Prone Mouse Model of Type 1 Diabetes (2011)
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Determinants of undercarboxylated and carboxylated osteocalcin concentrations in type 1 diabetes (2011)
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Distraction osteogenesis in TNF receptor 1 deficient mice is protected from chronic ethanol exposure (2011)
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Increasing duration of type 1 diabetes perturbs the strength–structure relationship and increases brittleness of bone (2010)
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Palmitate and insulin synergistically induce IL-6 expression in human monocytes (2010)
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