Hannah Barkley
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Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Hannah Barkley's research focuses on medical case studies, particularly concerning rare genetic disorders in infants. Her recent publications include work on profound hypotonia in newborns with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations, which is a form of porphyria. Barkley has also contributed to research on quality improvement training within pediatric residency programs and the development of evaluation systems in medical institutions. Her work involves collaborations with researchers at the University of Arkansas for Medical Sciences, including Carissa Rodriquez, Abhay A. Shukla, T. Burrow, and Alexis N. Roach, with whom she shares multiple publications.
Metrics
- Publications: 2
Selected Publications
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Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program (2025)
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Correction: Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria (2025)
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Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria (2024)
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04145 Profound hypotonia in a newborn with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations (2024)
Collaboration Network
Top Collaborators
- Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- 04145 Profound hypotonia in a newborn with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations
- Correction: Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- 04145 Profound hypotonia in a newborn with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations
- Correction: Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- 04145 Profound hypotonia in a newborn with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations
- Correction: Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- 04145 Profound hypotonia in a newborn with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations
- Correction: Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- Correction: Profound hypotonia in an infant with δ-aminolevulinic acid dehydratase deficient porphyria
- 04145 Profound hypotonia in a newborn with Biallelic δ-Aminolevulinic Acid Dehydratase (ALAD) mutations
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
- Creating a Roadmap for Longitudinal Quality Improvement Training in a Pediatric Residency Program
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