Sudhir V. Shah
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Also affiliated: Tulane University (1981–1992); University of Maryland, Baltimore (2007); United States Department of Veterans Affairs (1981–1987); Mayo Clinic (1978–1982); Sterling Hospitals (2022); Rashtrasant Tukadoji Maharaj Nagpur University (1985); University of British Columbia (2008); Innsbruck Medical University (2008); Taisho Pharmaceutical (Japan) (1991); Emory University (2010); Xavier University of Louisiana (1990); University of Pittsburgh (2008); University of Alabama (2008); Universität Innsbruck (2008); Zagazig University (1997); Washington University in St. Louis (1990); University of Arkansas Medical Center (1998–2021); University of Arkansas System (2025); Veterans Health Administration (1983); Tulane Medical Center (1984–1991); Leicester General Hospital (2008); United States Army Medical Research and Development Command (2002); University of Mississippi Medical Center (1996–1999); Ahmedabad Civil Hospital (2023); University of Alabama at Birmingham (2008); National Institute for Health and Care Research (2023); University of California San Diego (2008); Ege University (2007); Central Arkansas Veterans Healthcare System (2000–2025); Suamandeep Vidyapeeth University (2024); Southeast Louisiana Veterans Health Care System (1983–1987); Ospedale San Bortolo (2008); John L. McClellan Memorial Veterans Hospital (1992–2012); Ege Üniversitesi Tıp Fakültesi Hastanesi (2007); Smt. N.H.L. Municipal Medical College (2014–2022); Indiana University Indianapolis (2007–2008); University College London (2023); International Society of Nephrology (2007); Newcastle University (2023); Mississippi State University (1996)
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Biography and Research Information
OverviewAI-generated summary
Sudhir V. Shah's research interests encompass patient and public involvement in health sciences, with a particular focus on translating artificial intelligence into clinical practice and understanding behavioral science in healthcare. He has contributed to studies examining the burden of rare genetic disorders and the advancement of CRISPR/Cas9 technology for neurological disorders. His work also includes investigating the management of cardiovascular disease in kidney disease and assessing the utility of clinical exome sequencing for neurodevelopmental disorders.
Shah actively engages with various research initiatives, serving as a lay PPI representative for NIHR Applied Research Collaborations and as an advisory panellist for another ARC. He is a member of an NIHR Patient Safety Translation Research Centre, reviewing research studies with a focus on patient safety and behavioral science, such as statin reluctance. Additionally, he participates in UCL's Co-Production Group for Loneliness and Social Isolation and the Centre for Co-production in Health Research, advising on research funding. He also contributes to the Lived Experience Advisory Panel for Chronic Depression at East London NHS Foundation Trust, aiding in the development of patient and clinician tools.
His scholarly output includes a significant number of publications, reflected in an h-index of 68 and over 20,000 citations. He leads a research group and collaborates with colleagues at the University of Arkansas for Medical Sciences, including Manisha Singh, Oleg Karaduta, Sharda P. Singh, and Judit Megyesi.
Metrics
- h-index: 68
- Publications: 319
- Citations: 20,972
Selected Publications
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Effectiveness and efficiency of an add-on active outreach strategy on research enrollments in a drug intervention trial (2025)
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A simpler and more sensitive modified catalytic (bleomycin detectable) iron assay (2025)
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Iron Chelation Prevents Age‐Related Skeletal Muscle Sarcopenia in Klotho Gene Mutant Mice, a Genetic Model of Aging (2025)
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Ophthalmological Safety of Hydroxychloroquine in CKD (2023)
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Vascular Function Indices Are Strong Predictors of the Severity and Characteristics of Carotid Atherosclerosis (2021)
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Management of Cardiovascular Disease in Kidney Disease Study: Rationale and Design (2021)
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Autophagy Function and Regulation in Kidney Disease (2020)
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Home run—results of a chronic kidney disease Telemedicine Patient Education Study (2019)
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Effects of A Comprehensive Predialysis Education Program on the Home Dialysis Therapies: A Retrospective Cohort Study (2017)
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Carbamylated Low-Density Lipoprotein (cLDL)-Mediated Induction of Autophagy and Its Role in Endothelial Cell Injury (2016)
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Proteolytic processing and inactivation of CCL2/MCP-1 by meprins (2016)
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Autophagy in acute kidney injury (2016)
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Impact of Hydroxychloroquine on Atherosclerosis and Vascular Stiffness in the Presence of Chronic Kidney Disease (2015)
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Recent Advances in Understanding the Pathogenesis of Atherosclerosis in CKD Patients (2015)
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Evidence Suggesting a Role of Iron in a Mouse Model of Nephrogenic Systemic Fibrosis (2015)
Collaboration Network
Top Collaborators
- Autophagy in acute kidney injury
- Role and regulation of activation of caspases in cisplatin-induced injury to renal tubular epithelial cells
- Apoptotic mechanisms in acute renal failure
- p53-dependent Caspase-2 Activation in Mitochondrial Release of Apoptosis-inducing Factor and Its Role in Renal Tubular Epithelial Cell Injury
- Apoptotic pathways in ischemic acute renal failure
Showing 5 of 22 shared publications
- Carbamylated low-density lipoprotein induces death ofendothelial cells: A link to atherosclerosis in patients with kidney disease
- Apoptotic pathways in ischemic acute renal failure
- Chronic Uremia Stimulates LDL Carbamylation and Atherosclerosis
- Cisplatin Nephrotoxicity Is Mediated by Deoxyribonuclease I
- Ceramide synthase is essential for endonuclease-mediated death of renal tubular epithelial cells induced by hypoxia-reoxygenation
Showing 5 of 16 shared publications
- OXIDANT MECHANISMS IN TOXIC ACUTE RENAL FAILURE*
- In vitro and in vivo evidence suggesting a role for iron in cisplatin-induced nephrotoxicity
- Oxidants in Chronic Kidney Disease
- Oxidant mechanisms in toxic acute renal failure
- Increase in bleomycin-detectable iron in ischaemia/reperfusion injury to rat kidneys
Showing 5 of 12 shared publications
- Carbamylated low-density lipoprotein induces death ofendothelial cells: A link to atherosclerosis in patients with kidney disease
- Chronic Uremia Stimulates LDL Carbamylation and Atherosclerosis
- Cisplatin Nephrotoxicity Is Mediated by Deoxyribonuclease I
- Quantification of Carbamylated LDL in Human Sera by a New Sandwich ELISA
- Endonuclease G promotes cell death of non-invasive human breast cancer cells
Showing 5 of 12 shared publications
- OXIDANT MECHANISMS IN TOXIC ACUTE RENAL FAILURE*
- Oxidant mechanisms in toxic acute renal failure
- Oxidant Mechanisms in Gentamicin Nephrotoxicity
- Activation of a 15-kDa endonuclease in hypoxia/reoxygenation injury without morphologic features of apoptosis.
- An old enzyme with a new function: purification and characterization of a distinct matrix-degrading metalloproteinase in rat kidney cortex and its identification as meprin.
Showing 5 of 11 shared publications
- The Role of Iron in Diabetes and Its Complications
- New Insights into Nephrogenic Systemic Fibrosis
- Novel inflammatory mechanisms of accelerated atherosclerosis in kidney disease
- Novel approaches targeted toward oxidative stress for the treatment of chronic kidney disease
- Metal deposition in calcific uremic arteriolopathy
Showing 5 of 8 shared publications
- Apoptotic mechanisms in acute renal failure
- Role of cytochrome P-450 as a source of catalytic iron in cisplatin-induced nephrotoxicity
- Tubular cell damage in acute renal failure—apoptosis, necrosis, or both
- Role of caspases (ICE/CED 3 proteases) in DNA damage and cell death in response to a mitochondrial inhibitor, antimycin A Rapid Communication
- Role of enhanced ceramide generation in DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
Showing 5 of 8 shared publications
- OXIDANT MECHANISMS IN TOXIC ACUTE RENAL FAILURE*
- In vitro and in vivo evidence suggesting a role for iron in cisplatin-induced nephrotoxicity
- Oxidant mechanisms in toxic acute renal failure
- Ceramide synthase is essential for endonuclease-mediated death of renal tubular epithelial cells induced by hypoxia-reoxygenation
- Endonuclease induced DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
Showing 5 of 8 shared publications
- Effects of A Comprehensive Predialysis Education Program on the Home Dialysis Therapies: A Retrospective Cohort Study
- Impact of Hydroxychloroquine on Atherosclerosis and Vascular Stiffness in the Presence of Chronic Kidney Disease
- Home run—results of a chronic kidney disease Telemedicine Patient Education Study
- Recent Advances in Understanding the Pathogenesis of Atherosclerosis in CKD Patients
- Management of Cardiovascular Disease in Kidney Disease Study: Rationale and Design
Showing 5 of 8 shared publications
- Oxidants in Chronic Kidney Disease
- Effect of deferiprone, an oral iron chelator, in diabetic and non-diabetic glomerular disease
- Association of Catalytic Iron With Cardiovascular Disease
- The Role of Labile Iron in Kidney Disease and Treatment with Chelation
- Radiation Nephropathy is not Mitigated by Antagonists of Oxidative Stress
Showing 5 of 7 shared publications
- Endonuclease-induced DNA damage and cell death in oxidant injury to renal tubular epithelial cells.
- Increase in bleomycin-detectable iron in ischaemia/reperfusion injury to rat kidneys
- Activation of a 15-kDa endonuclease in hypoxia/reoxygenation injury without morphologic features of apoptosis.
- Role of reactive oxygen metabolites in DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
- Role of intracellular calcium in hydrogen peroxide-induced renal tubular cell injury
Showing 5 of 6 shared publications
- Impact of Hydroxychloroquine on Atherosclerosis and Vascular Stiffness in the Presence of Chronic Kidney Disease
- Carbamylated Low-Density Lipoprotein (cLDL)-Mediated Induction of Autophagy and Its Role in Endothelial Cell Injury
- Evidence Suggesting a Role of Iron in a Mouse Model of Nephrogenic Systemic Fibrosis
- Recent Advances in Understanding the Pathogenesis of Atherosclerosis in CKD Patients
- Gadolinium Contrast Agent-Induced CD163+ Ferroportin+ Osteogenic Cells in Nephrogenic Systemic Fibrosis
Showing 5 of 6 shared publications
- Role and regulation of activation of caspases in cisplatin-induced injury to renal tubular epithelial cells
- p53-dependent Caspase-2 Activation in Mitochondrial Release of Apoptosis-inducing Factor and Its Role in Renal Tubular Epithelial Cell Injury
- Transcriptional activation of caspase-6 and -7 genes by cisplatin-induced p53 and its functional significance in cisplatin nephrotoxicity
- Meprin A and meprin α generate biologically functional IL-1β from pro-IL-1β
- Regulation of caspase-3 and -9 activation in oxidant stress to RTE by forkhead transcription factors, Bcl-2 proteins, and MAP kinases
Showing 5 of 6 shared publications
- Carbamylated low-density lipoprotein induces death ofendothelial cells: A link to atherosclerosis in patients with kidney disease
- Quantification of Carbamylated LDL in Human Sera by a New Sandwich ELISA
- Carbamylated low‐density lipoprotein induces proliferation and increases adhesion molecule expression of human coronary artery smooth muscle cells
- Carbamylated-Oxidized LDL: Proatherosclerotic Effects on Endothelial Cells and Macrophages
- Carbamylated Low-Density Lipoprotein: Nontraditional Risk Factor for Cardiovascular Events in Patients With Chronic Kidney Disease
Showing 5 of 6 shared publications
- Transcriptional activation of caspase-6 and -7 genes by cisplatin-induced p53 and its functional significance in cisplatin nephrotoxicity
- Meprin A and meprin α generate biologically functional IL-1β from pro-IL-1β
- ADAM10 Is the Major Sheddase Responsible for the Release of Membrane-associated Meprin A
- Meprin A metalloproteinase and its role in acute kidney injury
- Proteolytic processing and inactivation of CCL2/MCP-1 by meprins
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