Alison R. Gouch
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Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Alison R. Gouch's research investigates the immune response to malaria, specifically focusing on the role of Interleukin-10 (IL-10) in controlling Plasmodium yoelii infections in mice. Her work has explored whether the early enhanced control of this infection in IL-10-deficient mice is dependent on other immune signaling molecules like Interferon-gamma (IFN-γ) and Interleukin-12 (IL-12), as well as the humoral immune response. Gouch's findings suggest that this enhanced control is independent of these factors. She has co-authored publications with Kara A. O’Neal and Sheldon L. Zeltner, both affiliated with the University of Arkansas for Medical Sciences, with whom she shares one publication.
Metrics
- Publications: 2
Selected Publications
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Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response (2026)
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Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response (2026)
Collaboration Network
Top Collaborators
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of Plasmodium yoelii infection in IL-10-deficient mice is independent of IFN-γ, IL-12, and the humoral response
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