Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Sheldon L. Zeltner studies the regulation of immune responses during parasitic infections. His research has focused on the role of Bhlhe40, a transcription factor, in controlling early interleukin-10 (IL-10) production from CD4+ T cells during infection with *Plasmodium yoelii* 17X. Zeltner has also investigated the implications of IL-10 deficiency in mice infected with *Plasmodium yoelii*, finding that early control of the infection in these mice is independent of interferon-gamma (IFN-γ), IL-12, and the humoral immune response. His work involves collaborations with researchers at the University of Arkansas for Medical Sciences, including Kara A. O’Neal, Jason S. Stumhofer, Camille L. Foscue, and Alison Gouch. Zeltner's scholarship metrics include an h-index of 1, with two total publications and five total citations.
Metrics
- h-index: 1
- Publications: 3
- Citations: 4
Selected Publications
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Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response (2026)
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Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection (2023)
Collaboration Network
Top Collaborators
- Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection
- Bhlhe40 limits early IL-10 production from CD4 <sup>+</sup> T cells during <i>Plasmodium yoelii</i> 17X infection
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
- Early enhanced control of <i>Plasmodium yoelii</i> infection in IL-10–deficient mice is independent of IFN-γ, IL-12, and the humoral response
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