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Biography and Research Information
OverviewAI-generated summary
Doug Wagner's research has focused on the immune response in animal models, particularly concerning microbial infections and host-pathogen interactions. His work has investigated the role of specific immune cells and cytokines in resistance to pathogens like *Listeria monocytogenes* and *Candida albicans*. Studies have explored the effects of probiotics on candidiasis in immunodeficient mice and examined candidiasis in mice with specific genetic knockouts, such as those for interferon and interleukin-10.
His research has also extended to aquatic animal health, with a publication on Edwardsiellosis in zebrafish. Wagner's scholarship metrics include an h-index of 23, with over 2,148 citations across 56 publications. He has collaborated with researchers at the National Center for Toxicological Research, including Anil K. Patri, Kuppan Gokulan, Mugimane G. Manjanatha, and Nathan A. Koonce, each sharing one publication with him.
Metrics
- h-index: 2
- Publications: 5
- Citations: 11
Selected Publications
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Biodistribution and toxic potential of silver nanoparticles when introduced to the female rat reproductive tract (2024)
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Intravaginal poly-(D, L-lactic-co-glycolic acid)-(polyethylene glycol) drug-delivery nanoparticles induce pro-inflammatory responses with Candida albicans infection in a mouse model (2020)
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Polyethylene glycol-functionalized poly (Lactic Acid-co-Glycolic Acid) and graphene oxide nanoparticles induce pro-inflammatory and apoptotic responses in Candida albicans-infected vaginal epithelial cells (2017)
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Probiotic bacteria prevent Salmonella – induced suppression of lymphoproliferation in mice by an immunomodulatory mechanism (2017)
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A metallo-β-lactamase is responsible for the degradation of ceftiofur by the bovine intestinal bacterium Bacillus cereus P41 (2014)
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Correction: Probiotic lactobacillus and estrogen effects on vaginal epithelial gene expression responses to Candida albicans (2012)
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Probiotic lactobacillus and estrogen effects on vaginal epithelial gene expression responses to Candida albicans (2012)
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Protection of Vaginal Epithelial Cells with Probiotic Lactobacilli and the Effect of Estrogen against Infection by Candida albicans (2012)
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Bovine Intestinal Bacteria Inactivate and Degrade Ceftiofur and Ceftriaxone with Multiple β-Lactamases (2011)
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Effects of Microbiota on GI Health: Gnotobiotic Research (2009)
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In Vitro Model of Colonization Resistance by the Enteric Microbiota: Effects of Antimicrobial Agents Used in Food-Producing Animals (2008)
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In Vitro Model of Colonization Resistance by the Enteric Microbiota: Effects of Antimicrobial Agents Used in Food-Producing Animals (2008)
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Probiotic bacteria are antagonistic to Salmonella enterica and Campylobacter jejuni and influence host lymphocyte responses in human microbiota‐associated immunodeficient and immunocompetent mice (2008)
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Vancomycin-Resistant Lactococcus lactis 1A-1 Isolated from a Competitive Exclusion Product Transfers Vancomycin Resistance Genes to Staphylococcus aureus (2008)
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Enhanced Production of Phospholipase C and Perfringolysin O (Alpha and Theta Toxins) in a Gatifloxacin-Resistant Strain of Clostridium perfringens (2007)
Collaboration Network
Top Collaborators
- Probiotic lactobacillus and estrogen effects on vaginal epithelial gene expression responses to Candida albicans
- Probiotic bacteria are antagonistic to Salmonella enterica and Campylobacter jejuni and influence host lymphocyte responses in human microbiota‐associated immunodeficient and immunocompetent mice
- Bovine Intestinal Bacteria Inactivate and Degrade Ceftiofur and Ceftriaxone with Multiple β-Lactamases
- Polyethylene glycol-functionalized poly (Lactic Acid-co-Glycolic Acid) and graphene oxide nanoparticles induce pro-inflammatory and apoptotic responses in Candida albicans-infected vaginal epithelial cells
- In Vitro Model of Colonization Resistance by the Enteric Microbiota: Effects of Antimicrobial Agents Used in Food-Producing Animals
Showing 5 of 12 shared publications
- Bovine Intestinal Bacteria Inactivate and Degrade Ceftiofur and Ceftriaxone with Multiple β-Lactamases
- Phenotypic and genotypic characterization of competitive exclusion products for use in poultry
- A metallo-β-lactamase is responsible for the degradation of ceftiofur by the bovine intestinal bacterium Bacillus cereus P41
- An In Vitro Assay To Evaluate Competitive Exclusion Products for Poultry
- In Vitro Model of Colonization Resistance by the Enteric Microbiota: Effects of Antimicrobial Agents Used in Food-Producing Animals
Showing 5 of 7 shared publications
- Early Resistance of Interleukin-10 Knockout Mice to Acute Systemic Candidiasis
- Potential hazards of probiotic bacteria for immunodeficient patients
- Risques potentiels dus aux bacteries probiotiques chez les patients immunodeficients
- Bovine Intestinal Bacteria Inactivate and Degrade Ceftiofur and Ceftriaxone with Multiple β-Lactamases
- A metallo-β-lactamase is responsible for the degradation of ceftiofur by the bovine intestinal bacterium Bacillus cereus P41
- Phenotypic and genotypic characterization of competitive exclusion products for use in poultry
- Vancomycin-Resistant Lactococcus lactis 1A-1 Isolated from a Competitive Exclusion Product Transfers Vancomycin Resistance Genes to Staphylococcus aureus
- Probiotic bacteria are antagonistic to Salmonella enterica and Campylobacter jejuni and influence host lymphocyte responses in human microbiota‐associated immunodeficient and immunocompetent mice
- Protection of Vaginal Epithelial Cells with Probiotic Lactobacilli and the Effect of Estrogen against Infection by Candida albicans
- Enhanced Production of Phospholipase C and Perfringolysin O (Alpha and Theta Toxins) in a Gatifloxacin-Resistant Strain of Clostridium perfringens
- Enhanced Production of Phospholipase C and Perfringolysin O (Alpha and Theta Toxins) in a Gatifloxacin-Resistant Strain of Clostridium perfringens
- Enhanced Production of Phospholipase C and Perfringolysin O (Alpha and Theta Toxins) in a Gatifloxacin-Resistant Strain of Clostridium perfringens
- Early Resistance of Interleukin-10 Knockout Mice to Acute Systemic Candidiasis
- Early Resistance of Interleukin-10 Knockout Mice to Acute Systemic Candidiasis
- Early Resistance of Interleukin-10 Knockout Mice to Acute Systemic Candidiasis
- A metallo-β-lactamase is responsible for the degradation of ceftiofur by the bovine intestinal bacterium Bacillus cereus P41
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