Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-08
M
Miguel A. B. Mercado
Ph.D.
Also affiliated: Cincinnati Children's Hospital Medical Center (2026); University of Cincinnati (2026)
4 h-index
12 pubs
44 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Miguel A. B. Mercado investigates host-pathogen interactions, focusing on the immune response to Chlamydia infections in mouse models. His research examines the roles of specific immune cells and signaling molecules in controlling the infection and preventing systemic dissemination and reproductive tract damage. Recent work has explored the necessity of innate versus adaptive T-cell-dependent Interferon-gamma (IFN-γ) production for systemic control, as well as the redundancy of CD4 T cell-dependent IFN-γ in the female reproductive tract.
Mercado's studies also investigate the function of antibodies in preventing systemic spread and the involvement of transcription factors like BHLHE40 in driving protective CD4 T cell responses. He collaborates with researchers at the University of Arkansas for Medical Sciences, including Lin-Xi Li, Yejin Kim, Rachel S. Palmer, and Priyangi A. Malaviarachchi, with whom he has co-authored multiple publications. His work contributes to understanding the complex immunological mechanisms underlying Chlamydia infections.
Metrics
- h-index: 4
- Publications: 12
- Citations: 44
Positions
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Ph.D. publications 2020–2026University of Arkansas for Medical Sciences Institution web page
Selected Publications
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CXCR6 marks polyfunctional effector CD4 T cells required for anti- Chlamydia immunity in the female reproductive tract (2026)
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The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284 (2025)
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BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2024)
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BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia (2023)
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Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice (2023)
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IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections (2022)
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IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections (2022)
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Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract (2020)
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Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract (2020)
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Antibody, but not B‐cell–dependent antigen presentation, plays an essential role in preventing Chlamydia systemic dissemination in mice (2020)
Collaboration Network
Top Collaborators
Lin‐Xi Li
University of Arkansas for Medical Sciences
10 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- Antibody, but not B‐cell–dependent antigen presentation, plays an essential role in preventing Chlamydia systemic dissemination in mice
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
Showing 5 of 10 shared publications
Priyangi A. Malaviarachchi
University of Arkansas for Medical Sciences
5 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- Antibody, but not B‐cell–dependent antigen presentation, plays an essential role in preventing Chlamydia systemic dissemination in mice
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Yejin Kim
University of Arkansas for Medical Sciences
5 shared publications
In database
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- The roles of TGFb signaling in CD4 T cell responses to Chlamydia infection in the female reproductive tract 9284
- CXCR6 marks polyfunctional effector CD4 T cells required for anti- Chlamydia immunity in the female reproductive tract
Wuying Du
University of Arkansas for Medical Sciences
4 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Jessica I. Gann
University of Arkansas for Medical Sciences
3 shared publications
In database
- Innate IFN-γ Is Essential for Systemic Chlamydia muridarum Control in Mice, While CD4 T Cell-Dependent IFN-γ Production Is Highly Redundant in the Female Reproductive Tract
- IFNγ and Antibody Synergize To Enhance Protective Immunity against Chlamydia Dissemination and Female Reproductive Tract Reinfections
- Innate IFNγ is essential for systemic Chlamydia control while CD4 T cell-dependent IFNγ production is highly redundant in the female reproductive tract
Charles M. Quick
University of Arkansas for Medical Sciences
3 shared publications
In database
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
Rachel S. Palmer
University of Arkansas for Medical Sciences
3 shared publications
In database
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
Lu Huang
University of Arkansas for Medical Sciences
3 shared publications
In database
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
Qiang Li
University of Arkansas for Medical Sciences (US)
2 shared publications
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
- BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia
Stephen J. McSorley
University of California, Davis (US)
1 shared publication
- Antibody, but not B‐cell–dependent antigen presentation, plays an essential role in preventing Chlamydia systemic dissemination in mice
Jessica A. Gann
University of Arkansas for Medical Sciences
1 shared publication
In database
- IFNγ and antibody synergize to enhance protective immunity against Chlamydia dissemination and female reproductive tract reinfections
Qiang Li
University of Arkansas for Medical Sciences (US)
1 shared publication
- Transcription factor Bhlhe40 plays a protective role during intravaginal Chlamydia muridarum infection in mice
Q Li
University of Arkansas for Medical Sciences
1 shared publication
In database
- CXCR6 marks polyfunctional effector CD4 T cells required for anti- Chlamydia immunity in the female reproductive tract
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