Hanan Hagar
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Also affiliated: Suez Canal University (2024); West Virginia University (2020); Zagazig University (1997–2026); Pulaski Technical College (2026); King Saud University (2002–2026); King Khalid University Hospital (2002–2023); John L. McClellan Memorial Veterans Hospital (1996–1997)
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Hanan Hagar's research investigates the protective effects of various compounds against induced organ damage in animal models, with a particular focus on oxidative stress and inflammation. Her work has explored the ameliorative properties of substances such as taurine, betaine, ghrelin, pyrrolidinedithiocarbamate, and Ginkgo biloba in protecting against injuries to the liver, kidneys, and gastrointestinal tract in rats. These studies often examine the underlying mechanisms, including the role of reactive oxygen species, glutathione levels, and inflammatory markers like Tumor Necrosis Factor-alpha.
Her research has been published in numerous peer-reviewed journals, contributing to the understanding of disease models and potential therapeutic interventions. Hagar's scholarly output is reflected in her h-index of 23 and over 1,655 citations across her 55 publications. She has demonstrated continued activity in research, with recent publications extending into the mid-2010s and a noted recent activity status.
Metrics
- h-index: 22
- Publications: 51
- Citations: 1,624
Selected Publications
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Dual-mechanism of phytochemicals in cancer therapy: synergistic anti-angiogenic potency in cancer and proangiogenic impact against chemotherapy-induced cytotoxicity in vital organs (2026)
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The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis (2024)
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Tyrosine phosphorylation in DNA damage and cell death in hypoxic injury to LLC-PK1 cells (1997)
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Role of reactive oxygen metabolites in DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells (1996)
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Endonuclease induced DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells (1996)
Collaboration Network
Top Collaborators
- Role of reactive oxygen metabolites in DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
- Endonuclease induced DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
- Tyrosine phosphorylation in DNA damage and cell death in hypoxic injury to LLC-PK1 cells
- Endonuclease induced DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
- Tyrosine phosphorylation in DNA damage and cell death in hypoxic injury to LLC-PK1 cells
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- Dual-mechanism of phytochemicals in cancer therapy: synergistic anti-angiogenic potency in cancer and proangiogenic impact against chemotherapy-induced cytotoxicity in vital organs
- Role of reactive oxygen metabolites in DNA damage and cell death in chemical hypoxic injury to LLC-PK1 cells
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- The risk of bone fractures in dementia patients receiving acetylcholinesterase inhibitors: a meta-analysis
- Dual-mechanism of phytochemicals in cancer therapy: synergistic anti-angiogenic potency in cancer and proangiogenic impact against chemotherapy-induced cytotoxicity in vital organs
- Dual-mechanism of phytochemicals in cancer therapy: synergistic anti-angiogenic potency in cancer and proangiogenic impact against chemotherapy-induced cytotoxicity in vital organs
- Dual-mechanism of phytochemicals in cancer therapy: synergistic anti-angiogenic potency in cancer and proangiogenic impact against chemotherapy-induced cytotoxicity in vital organs
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