Jungang Chen
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Also affiliated: Winthrop Rockefeller Foundation (2020)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Jungang Chen's research investigates the pathogenesis of Kaposi's Sarcoma-associated Herpesvirus (KSHV) and explores potential therapeutic targets. His work focuses on the role of Interleukin-1 family members and their signaling pathways in KSHV infection. Chen has also studied the impact of targeting specific proteins, such as BRD2 and BRD4, to inhibit the growth of KSHV-infected endothelial cells by suppressing LANA translation. Additionally, his research includes investigating the resistance mechanisms of primary effusion lymphoma cells to rapamycin and exploring the targeting of sphingosine kinase as a potential treatment for diffuse intrinsic pontine glioma. Chen has co-authored publications with collaborators Lu Dai and Zhiqiang Qin from the University of Arkansas for Medical Sciences.
Metrics
- h-index: 2
- Publications: 2
- Citations: 20
Selected Publications
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Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation (2026)
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Targeting Hyaluronan Signaling Overcomes Primary Effusion Lymphoma Cells Resistance to Rapamycin (2026)
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Oral Microbiota: Findings, Classification, Clinical Significance, and Therapeutic Interventions for Human Systemic Diseases (2026)
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Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis (2020)
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Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG) (2020)
Collaboration Network
Top Collaborators
- Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG)
- Targeting Hyaluronan Signaling Overcomes Primary Effusion Lymphoma Cells Resistance to Rapamycin
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG)
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG)
- Targeting Hyaluronan Signaling Overcomes Primary Effusion Lymphoma Cells Resistance to Rapamycin
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Targeting Hyaluronan Signaling Overcomes Primary Effusion Lymphoma Cells Resistance to Rapamycin
- Targeting BRD2 and BRD4 inhibit the growth of KSHV-infected immortalized endothelial cells through suppression of LANA translation
- Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG)
- Targeting Sphingosine Kinase by ABC294640 against Diffuse Intrinsic Pontine Glioma (DIPG)
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Role of Interleukin-1 Family Members and Signaling Pathways in KSHV Pathogenesis
- Oral Microbiota: Findings, Classification, Clinical Significance, and Therapeutic Interventions for Human Systemic Diseases
- Oral Microbiota: Findings, Classification, Clinical Significance, and Therapeutic Interventions for Human Systemic Diseases
- Oral Microbiota: Findings, Classification, Clinical Significance, and Therapeutic Interventions for Human Systemic Diseases
- Oral Microbiota: Findings, Classification, Clinical Significance, and Therapeutic Interventions for Human Systemic Diseases
- Oral Microbiota: Findings, Classification, Clinical Significance, and Therapeutic Interventions for Human Systemic Diseases
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