Luke Sultana
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Researcher
Graduate Student Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Luke Sultana's research focuses on developing and utilizing animal models to study osteogenesis imperfecta (OI), a genetic disorder affecting bone development. He has co-authored publications describing a new conditional knock-in mouse model for <i>Col1a1</i>, a gene involved in collagen production, which allows for the investigation of OI mechanisms. Sultana's work also explores the interplay between intrinsic lung defects and extrinsic skeletal issues in impaired lung function associated with OI, using these mouse models. His research collaborators at the University of Arkansas for Medical Sciences include Charles A. O’Brien, Roy Morello, and Milena Dimori. Sultana has contributed to two publications and has a total of three citations.
Metrics
- h-index: 1
- Publications: 2
- Citations: 3
Selected Publications
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A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta (2024)
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A New Mouse Model to Dissect the Contribution of Intrinsic Lung Defects Versus Extrinsic Skeletal Defects to Impaired Lung Function in Osteogenesis Imperfecta (2024)
Collaboration Network
Top Collaborators
- A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta
- A New Mouse Model to Dissect the Contribution of Intrinsic Lung Defects Versus Extrinsic Skeletal Defects to Impaired Lung Function in Osteogenesis Imperfecta
- A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta
- A New Mouse Model to Dissect the Contribution of Intrinsic Lung Defects Versus Extrinsic Skeletal Defects to Impaired Lung Function in Osteogenesis Imperfecta
- A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta
- A New Mouse Model to Dissect the Contribution of Intrinsic Lung Defects Versus Extrinsic Skeletal Defects to Impaired Lung Function in Osteogenesis Imperfecta
- A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta
- A New Mouse Model to Dissect the Contribution of Intrinsic Lung Defects Versus Extrinsic Skeletal Defects to Impaired Lung Function in Osteogenesis Imperfecta
- A New Mouse Model to Dissect the Contribution of Intrinsic Lung Defects Versus Extrinsic Skeletal Defects to Impaired Lung Function in Osteogenesis Imperfecta
- A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta
- A new <i>Col1a1</i> conditional knock-in mouse model to study osteogenesis imperfecta
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