Biography and Research Information
OverviewAI-generated summary
Samantha D. Crane's research focuses on host-pathogen interactions and disease models, particularly involving bacterial infections and their impact on mammalian systems. Her work has investigated the severity of SARS-CoV-2 infection in a mouse model of Down syndrome, examining heightened disease severity and mortality. Crane collaborates with researchers at the University of Arkansas for Medical Sciences, including Lu Huang, Zijing Zhang, Priyangi A. Malaviarachchi, and Hayley M. Theriot, contributing to a shared publication record. Her scholarship metrics include an h-index of 2, with 3 total publications and 26 total citations. Crane's active research status is indicated by her most recent publication in 2024.
Metrics
- h-index: 2
- Publications: 3
- Citations: 27
Positions
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Postdoctoral IRTA 2021–presentNational Institutes of Health Laboratory of Bacteriology ORCID
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Postdoctoral IRTA publications 2020–2024University of Arkansas for Medical Sciences ORCID
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Graduate Research Assistant 2016–2021University of Arkansas for Medical Sciences Microbiology and Immunology ORCID
Selected Publications
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SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome (2024)
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The Yersinia pestis GTPase BipA Promotes Pathogenesis of Primary Pneumonic Plague (2020)
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A Dual Role for the Plasminogen Activator Protease During the Preinflammatory Phase of Primary Pneumonic Plague (2020)
Collaboration Network
Top Collaborators
- A Dual Role for the Plasminogen Activator Protease During the Preinflammatory Phase of Primary Pneumonic Plague
- The Yersinia pestis GTPase BipA Promotes Pathogenesis of Primary Pneumonic Plague
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- A Dual Role for the Plasminogen Activator Protease During the Preinflammatory Phase of Primary Pneumonic Plague
- The Yersinia pestis GTPase BipA Promotes Pathogenesis of Primary Pneumonic Plague
- The Yersinia pestis GTPase BipA Promotes Pathogenesis of Primary Pneumonic Plague
- The Yersinia pestis GTPase BipA Promotes Pathogenesis of Primary Pneumonic Plague
- The Yersinia pestis GTPase BipA Promotes Pathogenesis of Primary Pneumonic Plague
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
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