Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
M
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Mara J. Campbell's research investigates the pathogenesis of *Staphylococcus aureus* infections, particularly osteomyelitis. Her work examines the role of bacterial factors, such as proteases and the *sarA* gene, in limiting virulence and influencing disease progression in animal models, specifically mice. Campbell also studies biofilm formation by *Staphylococcus aureus* and evaluates potential inhibitors. Additionally, her research includes the development of orthopedic implant materials, exploring cartilage-inspired surface textures for improved tribological performance. She has published 12 papers with 109 citations and an h-index of 6. Campbell collaborates with several researchers at the University of Arkansas for Medical Sciences, including Mark S. Smeltzer and Karen E. Beenken, with whom she shares multiple publications.
Metrics
- h-index: 6
- Publications: 12
- Citations: 114
Selected Publications
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Mechanistic insights into the pathogenesis and therapeutic recalcitrance of <i>Staphylococcus aureus</i> osteomyelitis (2026)
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Staphylococcus aureus Proteins Implicated in the Reduced Virulence of sarA and sarA/agr Mutants in Osteomyelitis (2025)
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The ability of <i>sarA</i> to limit protease production plays a key role in the pathogenesis of <i>Staphylococcus aureus</i> osteomyelitis irrespective of the functional status of <i>agr</i> (2024)
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RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis (2024)
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Increased production of aureolysin and staphopain A is a primary determinant of the reduced virulence of <i>Staphylococcus aureus sarA</i> mutants in osteomyelitis (2024)
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Comparative evaluation of small molecules reported to be inhibitors of <i>Staphylococcus aureus</i> biofilm formation (2023)
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The major role of <i>sarA</i> in limiting <i>Staphylococcus aureus</i> extracellular protease production <i>in vitro</i> is correlated with decreased virulence in diverse clinical isolates in osteomyelitis (2023)
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Cartilage-inspired surface textures for improved tribological performance of orthopedic implants (2022)
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The major role of <i>sarA</i> in limiting <i>Staphylococcus aureus</i> extracellular protease production is correlated with decreased virulence in diverse clinical isolates in osteomyelitis (2022)
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Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect (2021)
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Evaluation of a polymeric composite bone filler scaffold for local antibiotic delivery to prevent <i>Staphylococcus aureus</i> infection in a contaminated bone defect (2020)
Collaboration Network
Top Collaborators
Karen E. Beenken
University of Arkansas for Medical Sciences
8 shared publications
In database
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- The major role of <i>sarA</i> in limiting <i>Staphylococcus aureus</i> extracellular protease production <i>in vitro</i> is correlated with decreased virulence in diverse clinical isolates in osteomyelitis
- Increased production of aureolysin and staphopain A is a primary determinant of the reduced virulence of <i>Staphylococcus aureus sarA</i> mutants in osteomyelitis
- The ability of <i>sarA</i> to limit protease production plays a key role in the pathogenesis of <i>Staphylococcus aureus</i> osteomyelitis irrespective of the functional status of <i>agr</i>
Showing 5 of 8 shared publications
Mark S. Smeltzer
University of Arkansas for Medical Sciences
8 shared publications
In database
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- The major role of <i>sarA</i> in limiting <i>Staphylococcus aureus</i> extracellular protease production <i>in vitro</i> is correlated with decreased virulence in diverse clinical isolates in osteomyelitis
- Increased production of aureolysin and staphopain A is a primary determinant of the reduced virulence of <i>Staphylococcus aureus sarA</i> mutants in osteomyelitis
- The ability of <i>sarA</i> to limit protease production plays a key role in the pathogenesis of <i>Staphylococcus aureus</i> osteomyelitis irrespective of the functional status of <i>agr</i>
Showing 5 of 8 shared publications
Aura M. Ramírez
University of Arkansas for Medical Sciences (US)
4 shared publications
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
- The major role of <i>sarA</i> in limiting <i>Staphylococcus aureus</i> extracellular protease production <i>in vitro</i> is correlated with decreased virulence in diverse clinical isolates in osteomyelitis
- Increased production of aureolysin and staphopain A is a primary determinant of the reduced virulence of <i>Staphylococcus aureus sarA</i> mutants in osteomyelitis
- The major role of <i>sarA</i> in limiting <i>Staphylococcus aureus</i> extracellular protease production is correlated with decreased virulence in diverse clinical isolates in osteomyelitis
Karrar M. Alghazali
University of Arkansas at Little Rock (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Christopher M. Walker
University of Arkansas for Medical Sciences (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Bailey K. Jackson
University of Arkansas at Little Rock
1 shared publication
In database
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Christopher Griffin
University of Arkansas at Little Rock (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
William King
University of Arkansas at Little Rock
1 shared publication
In database
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Shawn E. Bourdo
University of Arkansas at Little Rock
1 shared publication
In database
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Rebecca E. Rifkin
University of Tennessee at Knoxville (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Silke Hecht
University of Tennessee at Knoxville (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Daniel G. Meeker
University of Arkansas for Medical Sciences (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
David E. Anderson
University of Tennessee at Knoxville (US)
1 shared publication
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Alexandru S. Biris
University of Arkansas at Little Rock
1 shared publication
In database
- Evaluation of a bone filler scaffold for local antibiotic delivery to prevent Staphylococcus aureus infection in a contaminated bone defect
Horace J. Spencer
University of Arkansas for Medical Sciences
1 shared publication
In database
- Comparative evaluation of small molecules reported to be inhibitors of <i>Staphylococcus aureus</i> biofilm formation
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