Mohammed S. Orloff
High Impact
Associate Professor
faculty
Epidemiology, College of Public Health
25 h-index
108 pubs
3,734 cited
Research Areas
Biography and Research Information
OverviewAI-generated summary
Mohammed S. Orloff is an Associate Professor in the Department of Epidemiology at the University of Arkansas for Medical Sciences. His research group focuses on public health issues, including the respiratory health impacts of electronic cigarette use, particularly among individuals who have never smoked conventional cigarettes. Dr. Orloff has also investigated the association of electronic cigarette use with conditions such as asthma, chronic obstructive pulmonary disease (COPD), and asthma-COPD overlap syndrome.
Further research by Dr. Orloff includes the temporal dynamics of SARS-CoV-2 genome and the detection of variants of concern in wastewater. He has also examined factors associated with smokefree home rules among Black/African American women smokers in rural communities and explored the role of paternal genetic variants in the risk of obstructive heart defects. His work has contributed to understanding changes in capacity building and sustained implementation among coalitions addressing racial/ethnic COVID-19 disparities.
With a career marked by extensive publication, Dr. Orloff has an h-index of 25 and over 3,700 citations across more than 100 publications. He frequently collaborates with researchers at the University of Arkansas for Medical Sciences, including Horacio Gómez-Acevedo, David W. Ussery, and Yasir Rahmatallah, as well as Ebrahim Jakoet from the University of Arkansas at Little Rock.
Metrics
- h-index: 25
- Publications: 108
- Citations: 3,734
Selected Publications
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Maternal Myo‐Inositol Intake and Congenital Heart Defects in Offspring: A Population‐Based Case–Control Study (2025)
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Abstract 4344025: Is Periconceptional Folic Acid Supplementation Associated with Cardiovascular Disease Risk Later in Life among Women who Delivered a Child with Congenital Heart Disease? (2025)
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A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior (2025)
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Capacity to address determinants of health among a social justice coalition in the United States (2025)
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Evidence-based practices are effective in increasing smoke-free home rules among Black women who smoke (2025)
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Exome Sequencing to Identify Novel Susceptibility Genes for Nonsyndromic Split‐Hand/Ft Malformation: A Report From the National Birth Defects Prevention Study (2025)
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Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers (2025)
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Home environment and cigarette quitting behaviors among rural Black/African American women caregivers. (2024)
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<scp>DNA</scp> methylation of the Lamin A/C gene is associated with congenital heart disease (2024)
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Changes in Capacity Building and Sustained Implementation Among a Statewide Coalition to Address Racial/Ethnic COVID-19 Disparities (2024)
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Tuberculosis and Risk of Emphysema among US Adults in the NHANES I Epidemiologic Follow-Up Study Cohort, 1971–1992 (2023)
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Tuberculosis and Risk of Emphysema among U.S. Adults in the NHANES I Epidemiologic Follow-Up Study Cohort, 1971 – 1992 (2023)
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Exploring the Relationship between Hog Farm Exposure and Chronic Obstructive Pulmonary Disease (COPD) (2022)
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Factors associated with smokefree rules in the homes of Black/African American women smokers residing in low-resource rural communities (2022)
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Electronic cigarette use and its association with asthma,chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers (2021)
Grants & Funding
- No FP attached UAMS Internal Research Awards Principal Investigator
- RFA-DD-18-001 Birth Defects Study To Evaluate Pregnancy exposures (BD-STEPS) II Core & Component B Steps -Stillbirth NIH Co-Investigator
- Birth Defects Study to Evaluate Pregnancy exposureS (BD-STEPS) Core? Arkansas Center and Stillbirth NIH Principal Investigator
- Center for Research, Health and Society NIH Co-Investigator
- Birth Defects Study to Evaluate Pregnancy exposureS (BD-STEPS) Core? Arkansas Center and Stillbirth NIH Co-Investigator
- Community Engagement Research Alliance (CEAL) 2023-2024 Renewal NIH/Nat. Heart, Lung & Blood Institute - Pass Through: Westat Principal Investigator
- Genes,Blood Pressure Variability and Cardiovascular Diseases NIH/Nat. Heart, Lung & Blood Institute Co-Investigator
- Arkansas Center for Health Disparities (ARCHD): An NIMHD COE NIH Co-Investigator
Collaboration Network
Top Collaborators
Jing Jin
16 shared publications
In database
- Evidence-based practices are effective in increasing smoke-free home rules among Black women who smoke
- Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S2 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Data from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Showing 5 of 16 shared publications
Horacio Gómez-Acevedo
16 shared publications
In database
- Exome Sequencing to Identify Novel Susceptibility Genes for Nonsyndromic Split‐Hand/Ft Malformation: A Report From the National Birth Defects Prevention Study
- Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S2 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Data from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Showing 5 of 16 shared publications
Yasir Rahmatallah
15 shared publications
In database
- Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S2 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Data from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S3 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Showing 5 of 15 shared publications
Yong‐Moon Park
15 shared publications
In database
- Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S2 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Data from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S3 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Showing 5 of 15 shared publications
David W. Ussery
15 shared publications
In database
- Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S2 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Data from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S3 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Showing 5 of 15 shared publications
Ebrahim Jakoet
14 shared publications
In database
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S2 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Data from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S3 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
- Supplementary Fig. S4 from A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Showing 5 of 14 shared publications
Shingo Ashida
12 shared publications
- Supplementary Figure 1B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Tables 1-3, Figure Legends 1-2 from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 1A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
Showing 5 of 12 shared publications
Gürkan Bebek
12 shared publications
- Supplementary Figure 1B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Tables 1-3, Figure Legends 1-2 from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 1A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
Showing 5 of 12 shared publications
Li Zhang
12 shared publications
- Supplementary Figure 1B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Tables 1-3, Figure Legends 1-2 from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 1A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
Showing 5 of 12 shared publications
Pan Zheng
12 shared publications
- Supplementary Figure 1B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Tables 1-3, Figure Legends 1-2 from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 1A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
Showing 5 of 12 shared publications
Donna M. Peehl
12 shared publications
- Supplementary Figure 1B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Tables 1-3, Figure Legends 1-2 from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 1A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
Showing 5 of 12 shared publications
Charis Eng
12 shared publications
- Supplementary Figure 1B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2B from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 2A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Tables 1-3, Figure Legends 1-2 from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
- Supplementary Figure 1A from Integrated Analysis Reveals Critical Genomic Regions in Prostate Tumor Microenvironment Associated with Clinicopathologic Phenotypes
Showing 5 of 12 shared publications
Pebbles Fagan
University of Arkansas for Medical Sciences
8 shared publications
In database
- Electronic cigarette use and its association with asthma,chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers
- Electronic cigarette use and its association with asthma,chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers
- Characterization of Electronic Cigarette Warning Statements Portrayed in YouTube Videos
- Factors associated with smokefree rules in the homes of Black/African American women smokers residing in low-resource rural communities
- Correction: Electronic cigarette use and its association with asthma, chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers
Showing 5 of 8 shared publications
Dina M. Jones
University of Arkansas for Medical Sciences
7 shared publications
In database
- Characterization of Electronic Cigarette Warning Statements Portrayed in YouTube Videos
- Factors associated with smokefree rules in the homes of Black/African American women smokers residing in low-resource rural communities
- Changes in Capacity Building and Sustained Implementation Among a Statewide Coalition to Address Racial/Ethnic COVID-19 Disparities
- Home environment and cigarette quitting behaviors among rural Black/African American women caregivers.
- Evidence-based practices are effective in increasing smoke-free home rules among Black women who smoke
Showing 5 of 7 shared publications
Emine Bircan
University of Arkansas for Medical Sciences
7 shared publications
In database
- Electronic cigarette use and its association with asthma,chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers
- Electronic cigarette use and its association with asthma,chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers
- Paternal genetic variants and risk of obstructive heart defects: A parent-of-origin approach
- Correction: Electronic cigarette use and its association with asthma, chronic obstructive pulmonary disease (COPD) and asthma-COPD overlap syndrome among never cigarette smokers
- Paternal genetic variants and risk of obstructive heart defects: A parent-of-origin approach
Showing 5 of 7 shared publications
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