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Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-15
H
Horacio Gómez-Acevedo
Associate Professor
Also affiliated: Arkansas Children's Hospital (2010–2015); University of Alberta (2004); University of Arkansas Medical Center (2014–2020); Arkansas Cardiology (2012); Stavros (2022); Arkansas Children's Nutrition Center (2011–2016); Arkansas Department of Agriculture (2013)
Faculty Researcher
Biomedical Informatics, College of Medicine
18 h-index
105 pubs
1,351 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Horacio Gómez-Acevedo's research focuses on understanding gene expression and its role in various biological processes, including bone metabolism and reproductive health. His work has investigated the impact of specific gene deletions and mutations on bone mass and resorption in animal models, such as mice and rats. He has also examined the effects of dietary factors, like soy formula, on reproductive outcomes in male piglets.
His publications also extend to human health, exploring the risks of adverse infant outcomes associated with maternal mental health and substance use disorders, as well as utilizing exome sequencing to identify genes associated with nonsyndromic split-hand/foot malformation. Gómez-Acevedo has a notable publication record with an h-index of 17 and over 1,300 citations, and he collaborates with researchers at the University of Arkansas for Medical Sciences and the University of Arkansas at Little Rock.
Metrics
- h-index: 18
- Publications: 105
- Citations: 1,351
Selected Publications
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MSR157 COMPARISON OF SAMPLING STRATEGIES TO ADDRESS SEVERE DATA IMBALANCE AND COMPUTATIONAL BENCHMARKING FOR TIME-TO-EVENT PREDICTIVE MODEL DEVELOPMENT ACROSS LOCAL AND HIGH-PERFORMANCE COMPUTING ENVIRONMENTS: PREDICTION OF ALCOHOL USE DISORDER AND OPIOID USE DISORDER AMONG ARKANSAS MEDICAL MARIJUANA CARDHOLDERS (2026)
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Abstract 7642: Nicotine dependence partially mediates the association between <i>IP6K3</i> genetic variation and risk of lung squamous cell carcinoma among smokers (2026)
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Deletion of the scavenger receptor Scarb1 in osteoblast progenitors and myeloid cells does not affect bone mass (2025)
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A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior (2025)
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MSR68 Development of a Discrete-Time-Updating Algorithm to Predict Cannabis Use Disorder Among Arkansas Medical Marijuana Cardholders (2025)
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MSR2 Developing a Feature Selection Workflow for Variable-Rich Data: A Case Study Utilizing Claims Data to Build Classifiers for the Prediction of Opioid Use Disorder Among Persons Authorized to Purchase Medical Cannabis (2025)
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MSR34 Development of a Novel Approach to Improve Binary Classification Prediction Tasks When Encountering Time-To-Event Data: Performance Comparison of Three Alternative Approaches To Predict Stimulant Use Disorder Among Persons Authorized To Purchase Medical Marijuana (2025)
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Deletion of the scavenger receptor <i>Scarb1</i> in osteoblast progenitors and myeloid cells does not affect bone mass (2025)
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An Activation Likelihood Estimation Meta-Analysis of Voxel-Based Morphometry Studies of Chemotherapy-Related Brain Volume Changes in Breast Cancer (2025)
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Exome Sequencing to Identify Novel Susceptibility Genes for Nonsyndromic Split‐Hand/Ft Malformation: A Report From the National Birth Defects Prevention Study (2025)
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Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers (2025)
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Fairness in Low Birthweight Predictive Models: Implications of Excluding Race/Ethnicity (2025)
Grants & Funding
As listed on this researcher's institutional profile.
- Birth Defects Study to Evaluate Pregnancy exposureS (BD-STEPS) Core? Arkansas Center and Stillbirth NIH Co-Investigator
- Center for Musculoskeletal Disease Research (CMDR) NIH/Nat. Inst. of General Medical Sciences Principal Investigator
- Tumor Shape Analysis and its Implications on Prognosis and Treatment TRI Translational Research Institute Principal Investigator
- RFA-DD-18-001 Birth Defects Study To Evaluate Pregnancy exposures (BD-STEPS) II Core & Component B Steps -Stillbirth NIH Co-Investigator
Collaboration Network
Top Collaborators
Ha‐Neui Kim
University of Arkansas for Medical Sciences
8 shared publications
In database
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
- Deletion of the Scavenger Receptor Scarb1 in Myeloid Cells Does Not Affect Bone Mass
Showing 5 of 8 shared publications
Stavros C. Manolagas
Central Arkansas Veterans Healthcare System
8 shared publications
In database
- Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass
- RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
Showing 5 of 8 shared publications
Michela Palmieri
Central Arkansas Veterans Healthcare System
7 shared publications
In database
- Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice
- Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass
- RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass
- Deletion of the Scavenger Receptor Scarb1 in Myeloid Cells Does Not Affect Bone Mass
- Retraction notice to “Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass” [Bone 170(2023) 116702]
Showing 5 of 7 shared publications
Teenamol E. Joseph
Central Arkansas Veterans Healthcare System
7 shared publications
In database
- Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice
- Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass
- RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass
- Deletion of the Scavenger Receptor Scarb1 in Myeloid Cells Does Not Affect Bone Mass
- Retraction notice to “Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass” [Bone 170(2023) 116702]
Showing 5 of 7 shared publications
Elena Ambrogini
University of Arkansas for Medical Sciences
7 shared publications
In database
- Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice
- Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass
- RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass
- Deletion of the Scavenger Receptor Scarb1 in Myeloid Cells Does Not Affect Bone Mass
- Retraction notice to “Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass” [Bone 170(2023) 116702]
Showing 5 of 7 shared publications
Charles A. O’Brien
Central Arkansas Veterans Healthcare System
6 shared publications
In database
- Deletion of the scavenger receptor Scarb1 in osteoblast progenitors does not affect bone mass
- RETRACTED: Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass
- Deletion of the Scavenger Receptor Scarb1 in Myeloid Cells Does Not Affect Bone Mass
- Retraction notice to “Deletion of the scavenger receptor Scarb1 in myeloid cells does not affect bone mass” [Bone 170(2023) 116702]
- Deletion of the scavenger receptor <i>Scarb1</i> in osteoblast progenitors and myeloid cells does not affect bone mass
Showing 5 of 6 shared publications
Intawat Nookaew
University of Arkansas for Medical Sciences
4 shared publications
In database
- Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Maria Almeida
University of Arkansas for Medical Sciences
4 shared publications
In database
- Neutralization of oxidized phospholipids attenuates age‐associated bone loss in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Filipa Ponte
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Aaron Warren
University of Arkansas for Medical Sciences
3 shared publications
In database
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Srividhya Iyer
University of Arkansas for Medical Sciences (US)
3 shared publications
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Erin M. Mannen
University of Arkansas for Medical Sciences (US)
3 shared publications
- Mmp13 deletion in mesenchymal cells increases bone mass and may attenuate the cortical bone loss caused by estrogen deficiency
- <i>Mmp-13</i> deletion in cells of the mesenchymal lineage increases bone mass, decreases endocortical osteoclast number, and attenuates the cortical bone loss caused by estrogen deficiency in mice
- Mmp13 deletion in mesenchymal cells increases bone mass and attenuates the cortical bone loss caused by estrogen deficiency
Mohammed S. Orloff
University of Arkansas for Medical Sciences
3 shared publications
In database
- Exome Sequencing to Identify Novel Susceptibility Genes for Nonsyndromic Split‐Hand/Ft Malformation: A Report From the National Birth Defects Prevention Study
- Abstract 1923: A tract of homozygosity analysis reveals methylation-driven <i>CSMD1</i> expression in non-small cell lung cancers
- A Tracts of Homozygosity Approach Identifies Methylation-Regulated <i>CSMD1</i> Expression Targets in Non–Small Cell Lung Cancers Related to Smoking Behavior
Allen M. Smith
University of Arkansas for Medical Sciences
3 shared publications
In database
- MSR34 Development of a Novel Approach to Improve Binary Classification Prediction Tasks When Encountering Time-To-Event Data: Performance Comparison of Three Alternative Approaches To Predict Stimulant Use Disorder Among Persons Authorized To Purchase Medical Marijuana
- MSR2 Developing a Feature Selection Workflow for Variable-Rich Data: A Case Study Utilizing Claims Data to Build Classifiers for the Prediction of Opioid Use Disorder Among Persons Authorized to Purchase Medical Cannabis
- MSR68 Development of a Discrete-Time-Updating Algorithm to Predict Cannabis Use Disorder Among Arkansas Medical Marijuana Cardholders
Corey J. Hayes
University of Arkansas for Medical Sciences
3 shared publications
In database
- MSR34 Development of a Novel Approach to Improve Binary Classification Prediction Tasks When Encountering Time-To-Event Data: Performance Comparison of Three Alternative Approaches To Predict Stimulant Use Disorder Among Persons Authorized To Purchase Medical Marijuana
- MSR2 Developing a Feature Selection Workflow for Variable-Rich Data: A Case Study Utilizing Claims Data to Build Classifiers for the Prediction of Opioid Use Disorder Among Persons Authorized to Purchase Medical Cannabis
- MSR68 Development of a Discrete-Time-Updating Algorithm to Predict Cannabis Use Disorder Among Arkansas Medical Marijuana Cardholders
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