Biography and Research Information
OverviewAI-generated summary
Oscar Zuniga's research focuses on the molecular mechanisms underlying cancer radioresistance, particularly in pancreatic and thyroid cancers. He has investigated signaling pathways involving RAS-RAF-miR-296-3p and its role in augmenting radioresistance through Rad18 expression. His work has identified the inhibitor of DNA binding 1 (ID1) as a potential marker for radioresistance in pancreatic cancer, utilizing genome-wide RNA sequencing for discovery. Zuniga has also explored methods to enhance radiosensitivity, such as the inhibition of Cyclin-dependent kinase 1 (CDK1). His research collaborations include work with Henrique Rodrigues, Lokesh Akana, Nükhet Aykin‐Burns, and Isabelle R. Miousse across institutions like the University of Arkansas for Medical Sciences and the University of Arkansas at Fayetteville. Zuniga has published 29 papers with an h-index of 12 and over 400 citations.
Metrics
- h-index: 2
- Publications: 7
- Citations: 9
Selected Publications
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Tracking domestication signals across populations of North American raccoons (Procyon lotor) via citizen science-driven image repositories (2025)
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Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction (2025)
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Abstract A011: Enhanced Radiosensitivity of Pancreatic Cancer Achieved through Inhibition of Cyclin-Dependent Kinase 1 (2024)
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Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1 (2024)
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RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers (2024)
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Abstract A006: Targeting SETD2 in combination with radiotherapy in rectal cancer (2024)
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Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers (2023)
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Abstract 2839: Methionine restriction radiosensitizes KRAS mutant rectal cancer (2023)
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Discovery of the inhibitor of DNA binding 1 as a novel marker for radioresistance in pancreatic cancer using genome-wide RNA-seq (2022)
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205 Targeting Homologous Repair to Overcome Genotoxic Therapy Resistance in Pancreatic Cancer (2022)
Collaboration Network
Top Collaborators
- Discovery of the inhibitor of DNA binding 1 as a novel marker for radioresistance in pancreatic cancer using genome-wide RNA-seq
- Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1
- RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers
- Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers
- 205 Targeting Homologous Repair to Overcome Genotoxic Therapy Resistance in Pancreatic Cancer
Showing 5 of 9 shared publications
- RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers
- Abstract 2839: Methionine restriction radiosensitizes KRAS mutant rectal cancer
- Abstract A006: Targeting SETD2 in combination with radiotherapy in rectal cancer
- Abstract A011: Enhanced Radiosensitivity of Pancreatic Cancer Achieved through Inhibition of Cyclin-Dependent Kinase 1
- Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction
- Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1
- Abstract A006: Targeting SETD2 in combination with radiotherapy in rectal cancer
- Abstract A011: Enhanced Radiosensitivity of Pancreatic Cancer Achieved through Inhibition of Cyclin-Dependent Kinase 1
- Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction
- Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1
- Abstract 2839: Methionine restriction radiosensitizes KRAS mutant rectal cancer
- Abstract A011: Enhanced Radiosensitivity of Pancreatic Cancer Achieved through Inhibition of Cyclin-Dependent Kinase 1
- Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1
- RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers
- Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers
- Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1
- Abstract A011: Enhanced Radiosensitivity of Pancreatic Cancer Achieved through Inhibition of Cyclin-Dependent Kinase 1
- Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction
- Abstract 2839: Methionine restriction radiosensitizes KRAS mutant rectal cancer
- Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction
- Abstract 2839: Methionine restriction radiosensitizes KRAS mutant rectal cancer
- Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction
- Abstract 2839: Methionine restriction radiosensitizes KRAS mutant rectal cancer
- Enhancing Rectal Cancer Radiosensitivity and Gut Protection through Methionine Restriction
- RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers
- Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers
- Enhanced radiosensitivity of pancreatic cancer achieved through inhibition of Cyclin-dependent kinase 1
- Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers
- RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers
- Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers
- RAS-RAF-miR-296-3p signaling axis increases Rad18 expression to augment radioresistance in pancreatic and thyroid cancers
- Ras-Raf-Mir-296-3p Signaling Axis Increases Rad18 Expression to Augment Radioresistance in Pancreatic and Thyroid Cancers
- 205 Targeting Homologous Repair to Overcome Genotoxic Therapy Resistance in Pancreatic Cancer
- 205 Targeting Homologous Repair to Overcome Genotoxic Therapy Resistance in Pancreatic Cancer
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