Shoon Lei Oo
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Researcher
Graduate Student Researcher
Research Areas
Biography and Research Information
OverviewAI-generated summary
Shoon Lei Oo's research focuses on therapeutic strategies for multiple myeloma and patient outcomes. Their work includes investigating the potential of G-protein coupled receptor class C group 5 member D (GPRC5D) therapies, as highlighted in a 2026 publication. Additionally, Oo has explored the influence of genomic and clinical high-risk features on patient responses to bispecific antibody therapy in relapsed/refractory multiple myeloma, also detailed in a 2026 publication. Another publication from 2025 addresses patient perspectives on the burden of straining in constipation. Oo collaborates with researchers at the University of Arkansas for Medical Sciences, including Stefan Longobardi, Carolina Schinke, Maurizio Zangari, and Frits van Rhee, with whom they have co-authored multiple publications.
Metrics
- Publications: 3
Selected Publications
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Insights and Future Directions of GPRC5D Therapies in Multiple Myeloma (2026)
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Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma. (2026)
Collaboration Network
Top Collaborators
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Insights and Future Directions of GPRC5D Therapies in Multiple Myeloma
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
- Influence of genomic and clinical high-risk features on outcomes after BCMA-directed bispecific antibody therapy in relapsed/refractory multiple myeloma.
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