Limited activity: This researcher has not published recently (last publication: 2013). Profile data may be outdated.
Match tier Confirmed
Presence Current · Arkansas
Last published 2013
Sources OpenAlex · ORCID
Refreshed 2026-10-05

Joe M. Fowler

Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.

Assistant Professor

NCTR

Also affiliated: United States Food and Drug Administration (2013)

1 h-index 8 pubs 4 cited

Research Areas

Links

ORCID Lab Website Research Group

OverviewAI-generated summary

Joe M. Fowler's research has focused on the evaluation of various substances for potential toxic effects. His work includes studying the genotoxicity of chemicals and the evaluation of their biological effects. Fowler has investigated the mechanisms of toxicity, particularly concerning DNA damage and cellular responses. His recent research has examined the effects of specific chemicals on biological systems, aiming to understand dose-response relationships and potential health risks.

Metrics

  • h-index: 1
  • Publications: 8
  • Citations: 4

Positions

  • Assistant Professor 2021–present
    Vanderbilt University Medical Center Medicine ORCID
  • Assistant Professor publications 2013
    National Center for Toxicological Research ORCID
  • Postdoctoral Researcher 2015–2021
    Vanderbilt University Medical Center Arrhythmia genetics research ORCID
  • Ph.D. Research (evolutionary genetics) 2008–2015
    University of California, Berkeley ORCID
  • Undergraduate research (developmental biology) 2005–2008
    Massachusetts Institute of Technology ORCID

Selected Publications

  • Study Protocol Preparation, Review, and Approval (2013)
    Elsevier eBooks DOI OpenAlex
  • Good Laboratory Practices (2013)
    Elsevier eBooks 1 citation DOI OpenAlex
  • Introduction (2013)
    Elsevier eBooks 1 citation DOI OpenAlex
  • Test Article (2013)
    Elsevier eBooks DOI OpenAlex
  • Final Report, Study Close-Out, and Conclusions (2013)
    Elsevier eBooks DOI OpenAlex

View all publications on OpenAlex →

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