J Panos
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Director of Translational Programs
Also affiliated: Northwestern University (2013); United States Food and Drug Administration (2014–2021); Western Michigan University (2008–2011); University of Wisconsin–Milwaukee (1997–1999)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
J Panos, Director of Translational Programs at the National Center for Toxicological Research, has a research focus that includes the study of diabetes complications and neurological disorders, such as Alzheimer's disease. Their work has explored the connection between Alzheimer's disease and type 3 diabetes, with a recent publication in 2021 examining this link in African American populations. Panos's scholarship metrics include an h-index of 12, with 58 total publications and 470 total citations. Key collaborators at the National Center for Toxicological Research include Sumit Sarkar and Daniel T. Sloper, with whom Panos has co-authored publications. Panos's recent activity indicates continued engagement in research.
Metrics
- h-index: 11
- Publications: 45
- Citations: 415
Positions
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Director of Translational ProgramsM-Lifesciences ORCID
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Director of Translational Programs publications 2014–2021National Center for Toxicological Research ORCID
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Director of Operations 2016–2018Spektron Systems ORCID
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ORISE Fellow 2012–2016National Center for Toxicological Research Neurotoxicology ORCID
Selected Publications
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Alzheimer’s disease: a step closer to understanding type 3 diabetes in African Americans (2021)
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Increased inflammation in BA21 brain tissue from African Americans with Alzheimer’s disease (2019)
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A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies (2019)
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Neurodegenerative Markers are Increased in Postmortem BA21 Tissue from African Americans with Alzheimer’s Disease (2017)
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P1‐330: Altered Levels of ABETA42 in BA21 Brain Region of African Americans Relative to Caucasians with Alzheimer's Disease (2016)
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Effects of perinatal methylphenidate (MPH) treatment in male and female Sprague–Dawley offspring (2014)
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Effects of developmental methylphenidate (MPH) treatment on monoamine neurochemistry of male and female rats (2014)
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Developmental methylphenidate (MPH) treatment in rats: Few effects on spatial memory (SM) in the Morris water (MWM) and Barnes mazes (BM) (2013)
Collaboration Network
Top Collaborators
- Increased inflammation in BA21 brain tissue from African Americans with Alzheimer’s disease
- Effects of perinatal methylphenidate (MPH) treatment in male and female Sprague–Dawley offspring
- Neurodegenerative Markers are Increased in Postmortem BA21 Tissue from African Americans with Alzheimer’s Disease
- Alzheimer’s disease: a step closer to understanding type 3 diabetes in African Americans
- Effects of developmental methylphenidate (MPH) treatment on monoamine neurochemistry of male and female rats
Showing 5 of 7 shared publications
- Increased inflammation in BA21 brain tissue from African Americans with Alzheimer’s disease
- Neurodegenerative Markers are Increased in Postmortem BA21 Tissue from African Americans with Alzheimer’s Disease
- Alzheimer’s disease: a step closer to understanding type 3 diabetes in African Americans
- P1‐330: Altered Levels of ABETA42 in BA21 Brain Region of African Americans Relative to Caucasians with Alzheimer's Disease
- Increased inflammation in BA21 brain tissue from African Americans with Alzheimer’s disease
- Neurodegenerative Markers are Increased in Postmortem BA21 Tissue from African Americans with Alzheimer’s Disease
- Alzheimer’s disease: a step closer to understanding type 3 diabetes in African Americans
- P1‐330: Altered Levels of ABETA42 in BA21 Brain Region of African Americans Relative to Caucasians with Alzheimer's Disease
- Effects of perinatal methylphenidate (MPH) treatment in male and female Sprague–Dawley offspring
- Developmental methylphenidate (MPH) treatment in rats: Few effects on spatial memory (SM) in the Morris water (MWM) and Barnes mazes (BM)
- Effects of developmental methylphenidate (MPH) treatment on monoamine neurochemistry of male and female rats
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- Effects of developmental methylphenidate (MPH) treatment on monoamine neurochemistry of male and female rats
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- Increased inflammation in BA21 brain tissue from African Americans with Alzheimer’s disease
- Alzheimer’s disease: a step closer to understanding type 3 diabetes in African Americans
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
- A Method for Sampling Rat Cerebrospinal Fluid with Minimal Blood Contamination: A Critical Tool for Biomarker Studies
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