Nicholas J. Baltz
Nephrology Fellow
Also affiliated: University of Arkansas Medical Center (2018); University of Florida (2020)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Nicholas J. Baltz, affiliated with the University of Arkansas for Medical Sciences, has a research profile indicated by a h-index of 3 and 5 total publications, with 15 citations. His work has involved collaboration with Joseph H. Holthoff, also at the University of Arkansas for Medical Sciences, on one shared publication. Baltz's recent publication in 2025, "The Second Slice Tells the Story: Evolving Cryoglobulinemia in Mesangial Glomerulonephritis," suggests an engagement with nephrology and immunology. The Medical Subject Headings associated with his work include "Animals, Humans, Mice, Hematopoiesis, Leukemia, Cell Line, Leukemia, Myeloid, Phosphorylation, Signal Transduction, Down-Regulation," pointing towards research in cellular and molecular mechanisms, potentially related to hematological malignancies and signal transduction pathways. His activity indicates recent engagement in research.
Metrics
- h-index: 3
- Publications: 5
- Citations: 17
Positions
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Nephrology Fellow publications 2016–2025University of Arkansas for Medical Sciences Institution web page
Selected Publications
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The Second Slice Tells the Story: Evolving Cryoglobulinemia in Mesangial Glomerulonephritis (2025)
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Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model (2020)
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PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia (2018)
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The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML (2016)
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JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency (2016)
Collaboration Network
Top Collaborators
- PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- The Dosage of Pten Is Critical in Determining the Disease Severity - a Mouse Model Differentially Mimicking JMML and CMML
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- JQ1, a Potential Therapeutic Molecule for Myeloid Leukemia with PTEN Deficiency
- PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia
- PTEN is indispensable for cells to respond to MAPK inhibitors in myeloid leukemia
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
- Sustained fetal hematopoiesis causes juvenile death from leukemia: evidence from a dual-age–specific mouse model
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