Dakota Redling
Researcher
Also affiliated: Arkansas Children's Hospital (2026); Oklahoma City University (2023); Arkansas Children's Nutrition Center (2026)
Unknown Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Dakota Redling's research focuses on understanding the mechanisms of chronic pain, particularly chemotherapy-induced peripheral neuropathy. Their work investigates how factors like myelin degradation and early-life stress contribute to the development and persistence of pain phenotypes, such as cold hypersensitivity. Redling has explored metabolic profiling in disease models to identify potential biomarkers and understand underlying physiological changes. Recent publications examine the impact of maternal care on offspring sensitivity and the effects of early-life stress on postsurgical pain outcomes in animal models. Collaborations include work with Kayleigh Amber Rodriguez, Laura Osborn, and Mary Grace Bishop at the University of Arkansas for Medical Sciences, with whom Redling has co-authored multiple publications.
Metrics
- h-index: 1
- Publications: 7
- Citations: 2
Selected Publications
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Degraded myelin is associated with cold hypersensitivity in paclitaxel-induced peripheral neuropathy (2026)
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Impact of Early Life Stress on the Composition of Gut Microbiome (2026)
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A Novel Model of Paclitaxel-Induced Peripheral Neuropathy Produces aClinically Relevant Phenotype in Mice (2026)
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Impact of Maternal Care Behaviors on Mechanical Sensitivity of Adult Offspring (2025)
Collaboration Network
Top Collaborators
- A Novel Model of Paclitaxel-Induced Peripheral Neuropathy Produces aClinically Relevant Phenotype in Mice
- Impact of Maternal Care Behaviors on Mechanical Sensitivity of Adult Offspring
- Degraded myelin is associated with cold hypersensitivity in paclitaxel-induced peripheral neuropathy
- A Novel Model of Paclitaxel-Induced Peripheral Neuropathy Produces aClinically Relevant Phenotype in Mice
- Impact of Maternal Care Behaviors on Mechanical Sensitivity of Adult Offspring
- Degraded myelin is associated with cold hypersensitivity in paclitaxel-induced peripheral neuropathy
- A Novel Model of Paclitaxel-Induced Peripheral Neuropathy Produces aClinically Relevant Phenotype in Mice
- Impact of Maternal Care Behaviors on Mechanical Sensitivity of Adult Offspring
- Degraded myelin is associated with cold hypersensitivity in paclitaxel-induced peripheral neuropathy
- A Novel Model of Paclitaxel-Induced Peripheral Neuropathy Produces aClinically Relevant Phenotype in Mice
- Impact of Maternal Care Behaviors on Mechanical Sensitivity of Adult Offspring
- A Novel Model of Paclitaxel-Induced Peripheral Neuropathy Produces aClinically Relevant Phenotype in Mice
- Degraded myelin is associated with cold hypersensitivity in paclitaxel-induced peripheral neuropathy
- Degraded myelin is associated with cold hypersensitivity in paclitaxel-induced peripheral neuropathy
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