Michel Fausther
Diagnostic and Molecular Pathology Resident
Also affiliated: University of Arkansas Medical Center (2011–2018); Yale University (2011–2012); Centre hospitalier universitaire de Québec (2008–2014); Central Arkansas Veterans Healthcare System (2013–2017); Centre hospitalier de l'Université Laval (2009–2017); Université Laval (2005–2017); Centre de recherche du CHU de Québec-Université Laval (2014–2017); CHU de Québec-Université Laval (2011–2013); Université du Québec à Trois-Rivières (2004)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Michel Fausther's research has focused on cancer-related molecular mechanisms, particularly investigating the role of specific molecular pathways in disease progression. Their work has contributed to understanding cancer genomics and diagnostics, with a publication in 2019 detailing findings in this area. Fausther's contributions are situated within the broader field of health and medical research impacts.
Metrics
- h-index: 23
- Publications: 71
- Citations: 1,456
Positions
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Diagnostic and Molecular Pathology Resident 2023–presentCHU de Québec Medicine ORCID
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Diagnostic and Molecular Pathology Resident publications 2010–2019University of Arkansas for Medical Sciences ORCID
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Assistant Professor 2015–2017University of Arkansas for Medical Sciences Internal Medicine - GI Division ORCID
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Research Instructor 2013–2015University of Arkansas for Medical Sciences Internal Medicine · Gastroenterology and Hepatology Division ORCID
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Post-Doctoral Fellow 2011–2013University of Arkansas for Medical Sciences Internal Medicine - Gastroenterology and Hepatology Division ORCID
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Post-Doctoral Associate 2010–2011Yale University Yale Liver Center - Digestive Diseases Section ORCID
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Student Teaching Assistant 2000–2003Université du Québec à Trois-Rivières Département de Chimie-Biologie ORCID
Selected Publications
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Reduction in SNAP-23 Alters Microfilament Organization in Myofibrobastic Hepatic Stellate Cells (2019)
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Junctional adhesion molecules JAM-B and JAM-C promote autoimmune-mediated liver fibrosis in mice (2018)
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Extracellular adenosine: a critical signal in liver fibrosis (2018)
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The Cholangiocyte Adenosine‐IL-6 Axis Regulates Survival During Biliary Cirrhosis (2017)
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Liver myofibroblasts of murine origins express mesothelin: Identification of novel rat mesothelin splice variants* (2017)
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An Elf2-like transcription factor acts as repressor of the mouse ecto-5′-nucleotidase gene expression in hepatic myofibroblasts (2017)
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Generation and characterization of polyclonal and monoclonal antibodies to human NTPDase2 including a blocking antibody (2017)
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Transforming Growth Factors α and β Are Essential for Modeling Cholangiocarcinoma Desmoplasia and Progression in a Three-Dimensional Organotypic Culture Model (2017)
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Generation and Characterization of Specific Antibodies to the Murine and Human Ectonucleotidase NTPDase8 (2017)
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Contribution of Liver Nonparenchymal Cells to Hepatic Fibrosis: Interactions with the Local Microenvironment (2017)
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Extracellular vesicles carry microRNA‐195 to intrahepatic cholangiocarcinoma and improve survival in a rat model (2016)
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Peer Review Report For: I drink for my liver, Doc: emerging evidence that coffee prevents cirrhosis [version 1; peer review: 3 approved] (2015)
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Peer Review Report For: I drink for my liver, Doc: emerging evidence that coffee prevents cirrhosis [version 1; peer review: 3 approved] (2015)
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I drink for my liver, Doc: emerging evidence that coffee prevents cirrhosis (2015)
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I drink for my liver, Doc: emerging evidence that coffee prevents cirrhosis (2015)
Collaboration Network
Top Collaborators
- Extracellular vesicles carry microRNA‐195 to intrahepatic cholangiocarcinoma and improve survival in a rat model
- Advances in cholangiocyte immunobiology
- Coexpression of ecto-5′-nucleotidase/CD73 with specific NTPDases differentially regulates adenosine formation in the rat liver
- Activated hepatic stellate cells upregulate transcription of ecto-5′-nucleotidase/CD73 via specific SP1 and SMAD promoter elements
- Contribution of Myofibroblasts of Different Origins to Liver Fibrosis
Showing 5 of 46 shared publications
- Contribution of Myofibroblasts of Different Origins to Liver Fibrosis
- How does coffee prevent liver fibrosis? biological plausibility for recent epidemiological observations
- Establishment and Characterization of Rat Portal Myofibroblast Cell Lines
- Pathological Changes in Pulmonary Circulation in Carbon Tetrachloride (ccl4)-Induced Cirrhotic Mice
- NT5E Mutations That Cause Human Disease Are Associated with Intracellular Mistrafficking of NT5E Protein
Showing 5 of 20 shared publications
- Establishment and Characterization of Rat Portal Myofibroblast Cell Lines
- Pathological Changes in Pulmonary Circulation in Carbon Tetrachloride (ccl4)-Induced Cirrhotic Mice
- NT5E Mutations That Cause Human Disease Are Associated with Intracellular Mistrafficking of NT5E Protein
- Expression of mediators of purinergic signaling in human liver cell lines
- The Cholangiocyte Adenosine‐IL-6 Axis Regulates Survival During Biliary Cirrhosis
Showing 5 of 8 shared publications
- Coexpression of ecto-5′-nucleotidase/CD73 with specific NTPDases differentially regulates adenosine formation in the rat liver
- Establishment and Characterization of Rat Portal Myofibroblast Cell Lines
- Generation and Characterization of Specific Antibodies to the Murine and Human Ectonucleotidase NTPDase8
- Generation and characterization of polyclonal and monoclonal antibodies to human NTPDase2 including a blocking antibody
- Coexpression of ecto-5′-nucleotidase/CD73 with specific NTPDases differentially regulates adenosine formation in the rat liver
- Activated hepatic stellate cells upregulate transcription of ecto-5′-nucleotidase/CD73 via specific SP1 and SMAD promoter elements
- Posttranslational regulation of tissue inhibitor of metalloproteinase-1 by calcium-dependent vesicular exocytosis
- CXCL12 induces hepatic stellate cell contraction through a calcium-independent pathway
- Posttranslational regulation of tissue inhibitor of metalloproteinase-1 by calcium-dependent vesicular exocytosis
- MCP-1 downregulates MMP-9 export via vesicular redistribution to lysosomes in rat portal fibroblasts
- Coexpression of ecto-5′-nucleotidase/CD73 with specific NTPDases differentially regulates adenosine formation in the rat liver
- Generation and Characterization of Specific Antibodies to the Murine and Human Ectonucleotidase NTPDase8
- Generation and characterization of polyclonal and monoclonal antibodies to human NTPDase2 including a blocking antibody
- How does coffee prevent liver fibrosis? biological plausibility for recent epidemiological observations
- I drink for my liver, Doc: emerging evidence that coffee prevents cirrhosis
- I drink for my liver, Doc: emerging evidence that coffee prevents cirrhosis
- Role of purinergic P2X receptors in the control of liver homeostasis
- Erratum: Role of purinergic P2X receptors in the control of liver homeostasis
- Activated hepatic stellate cells upregulate transcription of ecto-5′-nucleotidase/CD73 via specific SP1 and SMAD promoter elements
- CXCL12 induces hepatic stellate cell contraction through a calcium-independent pathway
- Activated hepatic stellate cells upregulate transcription of ecto-5′-nucleotidase/CD73 via specific SP1 and SMAD promoter elements
- CXCL12 induces hepatic stellate cell contraction through a calcium-independent pathway
- NT5E Mutations That Cause Human Disease Are Associated with Intracellular Mistrafficking of NT5E Protein
- Posttranslational regulation of tissue inhibitor of metalloproteinase-1 by calcium-dependent vesicular exocytosis
- Advances in cholangiocyte immunobiology
- MCP-1 downregulates MMP-9 export via vesicular redistribution to lysosomes in rat portal fibroblasts
- Coexpression of ecto-5′-nucleotidase/CD73 with specific NTPDases differentially regulates adenosine formation in the rat liver
- Generation and Characterization of Specific Antibodies to the Murine and Human Ectonucleotidase NTPDase8
- Extracellular vesicles carry microRNA‐195 to intrahepatic cholangiocarcinoma and improve survival in a rat model
- Transforming Growth Factors α and β Are Essential for Modeling Cholangiocarcinoma Desmoplasia and Progression in a Three-Dimensional Organotypic Culture Model
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