Giulia Baldini Institution-verified
Sourced from institutional research profiles (UAMS TRI or ARA).
Researcher
faculty
Research Areas
Biography and Research Information
OverviewAI-generated summary
Giulia Baldini, a faculty member at the University of Arkansas for Medical Sciences, leads a research group investigating cellular mechanisms relevant to metabolic disorders and cancer. Her work includes studies on the role of Liraglutide in counteracting endoplasmic reticulum stress in hypothalamic neurons, as well as research into the localization of MC4R in these neurons. Baldini also investigates cell death pathways in acute lymphoblastic leukemia, specifically examining the susceptibility of leukemia cells to microtubule depolymerization during different cell cycle phases. Her scholarship metrics include an h-index of 27, with 59 total publications and over 5,800 citations, designating her as a highly cited researcher. She has collaborated with researchers at the University of Arkansas at Fayetteville and within the University of Arkansas for Medical Sciences, including Kevin D. Phelan, Haven Griffin, Kevin D. Raney, and Randall R Rainwater.
Metrics
- h-index: 28
- Publications: 59
- Citations: 5,910
Selected Publications
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MC4R Localizes at Excitatory Postsynaptic and Peri-Postsynaptic Sites of Hypothalamic Neurons in Primary Culture (2024)
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Liraglutide Counteracts Endoplasmic Reticulum Stress in Palmitate-Treated Hypothalamic Neurons without Restoring Mitochondrial Homeostasis (2022)
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Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways (2022)
Grants & Funding
Collaboration Network
Top Collaborators
- Liraglutide Counteracts Endoplasmic Reticulum Stress in Palmitate-Treated Hypothalamic Neurons without Restoring Mitochondrial Homeostasis
- MC4R Localizes at Excitatory Postsynaptic and Peri-Postsynaptic Sites of Hypothalamic Neurons in Primary Culture
- Liraglutide Counteracts Endoplasmic Reticulum Stress in Palmitate-Treated Hypothalamic Neurons without Restoring Mitochondrial Homeostasis
- MC4R Localizes at Excitatory Postsynaptic and Peri-Postsynaptic Sites of Hypothalamic Neurons in Primary Culture
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- Primary acute lymphoblastic leukemia cells are susceptible to microtubule depolymerization in G1 and M phases through distinct cell death pathways
- A Novel Tactile Device for Safe Human-Robot Interaction in Industrial Scenarios
- A Novel Tactile Device for Safe Human-Robot Interaction in Industrial Scenarios
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