Md. Mahbub Hasan
Researcher
Also affiliated: Khulna University of Engineering and Technology (2025); George Mason University (2023); Dhaka University of Engineering & Technology (2021–2026); Bangladesh University of Professionals (2017)
Unknown Researcher
Research Areas
Biography and Research Information
OverviewAI-generated summary
Md. Mahbub Hasan's research focuses on molecular mechanisms underlying disease and the development of therapeutic interventions. His work includes investigating oncogenic splicing events, specifically targeting the <i>EZH2</i> gene and its regulatory network for potential cancer therapies. He also studies the role of splicing factors SRSF6 and SRSF1 in generating Wnt-sensitive isoforms of the MuSK protein, which is relevant to neuromuscular disorders. Hasan's research extends to nanotechnological approaches for ocular drug delivery systems aimed at treating eye diseases. Additionally, his work has explored the application of genetic algorithms for optimization problems, as evidenced by his publication on a time-varying mutation operator. Hasan has a h-index of 4 with 112 citations across 7 publications and collaborates with researchers at the University of Arkansas for Medical Sciences, including Preeti Nagar and Mohammad Alinoor Rahman.
Metrics
- h-index: 4
- Publications: 8
- Citations: 126
Selected Publications
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Targeting <i>EZH2</i> oncogenic splicing: decoding the regulatory network and antisense correction (2026)
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CRISPR-mediated Correction of Oncogenic AS-NMD in Splicing Factor Mutant Cancer (2026)
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Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction (2026)
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SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform (2025)
Collaboration Network
Top Collaborators
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- SRSF6 and SRSF1 coordinately enhance the inclusion of human <i>MUSK</i> exon 10 to generate a Wnt-sensitive MuSK isoform
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
- Targeting <i>EZH2</i> Oncogenic Splicing: Decoding the Regulatory Network and Antisense Correction
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