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Listed
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-10-06
J
Judit K. Megyesi
High Impact
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Also affiliated: The University of Texas MD Anderson Cancer Center (2013); University of Arizona (2019); Medical University of South Carolina (2019); University of Arkansas Medical Center (1999–2020); Massachusetts General Hospital (2013); Southern Arizona VA Health Care System (2019); Central Arkansas Veterans Healthcare System (2002–2022); Saitama International Medical Center (2013); John L. McClellan Memorial Veterans Hospital (2002–2019); The University of Texas Medical Branch at Galveston (1995–2008); The University of Tokyo (2004); Saitama Medical University (2013); The University of Osaka (2004)
40 h-index
78 pubs
5,065 cited
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Judit Megyesi's research investigates mechanisms of organ injury and protection, with a particular focus on kidney and muscle tissue. Her work has explored how specific deficiencies or interventions can alter the course of disease. For instance, she has published on how neutral ceramidase deficiency offers protection against cisplatin-induced acute kidney injury in mice. Additionally, her research has examined the impact of iron chelation in preventing age-related skeletal muscle sarcopenia in a genetic mouse model of aging. She has also investigated the protective effects of γ-tocotrienol against mitochondrial dysfunction and renal function decline following ischemia.
Megyesi's scholarly contributions are reflected in her metrics, including an h-index of 40 and over 5,000 citations from 77 publications. She has collaborated with researchers at the University of Arkansas for Medical Sciences, including Oleg Karaduta, Sharda P. Singh, Sudhir V. Shah, and Chhanda Bose, on shared publications. Her recent activity indicates an ongoing engagement with her research areas.
Metrics
- h-index: 40
- Publications: 78
- Citations: 5,065
Selected Publications
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Iron Chelation Prevents Age‐Related Skeletal Muscle Sarcopenia in Klotho Gene Mutant Mice, a Genetic Model of Aging (2025)
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Neutral ceramidase deficiency protects against cisplatin-induced acute kidney injury (2022)
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γ-Tocotrienol Protects against Mitochondrial Dysfunction, Energy Deficits, Morphological Damage, and Decreases in Renal Functions after Renal Ischemia (2021)
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C57BL/6 mice require a higher dose of cisplatin to induce renal fibrosis and CCL2 correlates with cisplatin-induced kidney injury (2020)
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Deletion of VDAC1 Hinders Recovery of Mitochondrial and Renal Functions After Acute Kidney Injury (2020)
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Protein kinase Cα mediates recovery of renal and mitochondrial functions following acute injury (2019)
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Complement C1r serine protease contributes to kidney fibrosis (2019)
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Proximal Tubule β2-Adrenergic Receptor Mediates Formoterol-Induced Recovery of Mitochondrial and Renal Function after Ischemia-Reperfusion Injury (2019)
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5-HT1F receptor regulates mitochondrial homeostasis and its loss potentiates acute kidney injury and impairs renal recovery (2018)
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Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease (2018)
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Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury (2017)
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Pericytes and immune cells contribute to complement activation in tubulointerstitial fibrosis (2017)
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Suramin Protects From Cisplatin‐Induced Acute Kidney Injury (2016)
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Deletion of protein kinase C-ε attenuates mitochondrial dysfunction and ameliorates ischemic renal injury (2016)
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Cdk2 phosphorylation of Bcl-xL after stress converts it to a pro-apoptotic protein mimicking Bax/Bak (2016)
Collaboration Network
Top Collaborators
Robert L. Safirstein
University of Arkansas for Medical Sciences (US)
16 shared publications
- The cell cycle and acute kidney injury
- Metabolomic study of cisplatin-induced nephrotoxicity
- Positive effect of the induction of p21WAF1/CIP1 on the course of ischemic acute renal failure
- Protection of renal cells from cisplatin toxicity by cell cycle inhibitors
- The lack of a functional p21 WAF1/CIP1 gene ameliorates progression to chronic renal failure
Showing 5 of 16 shared publications
Peter M. Price
University of Arkansas for Medical Sciences (US)
15 shared publications
- Activation and involvement of p53 in cisplatin-induced nephrotoxicity
- The cell cycle and acute kidney injury
- Alterations of PPARα and its coactivator PGC-1 in cisplatin-induced acute renal failure
- Protection of renal cells from cisplatin toxicity by cell cycle inhibitors
- The lack of a functional p21 WAF1/CIP1 gene ameliorates progression to chronic renal failure
Showing 5 of 15 shared publications
Didier Portilla
Central Arkansas Veterans Healthcare System (US)
11 shared publications
- Metabolomic study of cisplatin-induced nephrotoxicity
- Alterations of PPARα and its coactivator PGC-1 in cisplatin-induced acute renal failure
- Transgenic expression of proximal tubule peroxisome proliferator–activated receptor-α in mice confers protection during acute kidney injury
- PPAR-α ligand ameliorates acute renal failure by reducing cisplatin-induced increased expression of renal endonuclease G
- Proximal tubule PPARα attenuates renal fibrosis and inflammation caused by unilateral ureteral obstruction
Showing 5 of 11 shared publications
Leah J. Siskind
University of Louisville (US)
8 shared publications
- Repeated administration of low-dose cisplatin in mice induces fibrosis
- Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- C57BL/6 mice require a higher dose of cisplatin to induce renal fibrosis and CCL2 correlates with cisplatin-induced kidney injury
- Suramin protects from cisplatin-induced acute kidney injury
Showing 5 of 8 shared publications
Adel Tarcsafalvi
University of Arkansas for Medical Sciences (US)
7 shared publications
- Gender differences control the susceptibility to ER stress-induced acute kidney injury
- Increased expression of p21WAF1/CIP1 in kidney proximal tubules mediates fibrosis
- Cdk2-dependent phosphorylation of p21 regulates the role of Cdk2 in cisplatin cytotoxicity
- INTRAHEPATIC EXPRESSION AND RELEASE OF VASCULAR ENDOTHELIAL GROWTH FACTOR FOLLOWING ORTHOTOPIC LIVER TRANSPLANTATION IN THE RAT
- Cdk2 phosphorylation of Bcl-xL after stress converts it to a pro-apoptotic protein mimicking Bax/Bak
Showing 5 of 7 shared publications
Mark A. Doll
University of Louisville (US)
7 shared publications
- Repeated administration of low-dose cisplatin in mice induces fibrosis
- Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- C57BL/6 mice require a higher dose of cisplatin to induce renal fibrosis and CCL2 correlates with cisplatin-induced kidney injury
- Suramin protects from cisplatin-induced acute kidney injury
Showing 5 of 7 shared publications
Cierra N. Sharp
University of Louisville (US)
7 shared publications
- Repeated administration of low-dose cisplatin in mice induces fibrosis
- Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- C57BL/6 mice require a higher dose of cisplatin to induce renal fibrosis and CCL2 correlates with cisplatin-induced kidney injury
- Suramin protects from cisplatin-induced acute kidney injury
Showing 5 of 7 shared publications
Levi J. Beverly
University of Louisville (US)
7 shared publications
- Repeated administration of low-dose cisplatin in mice induces fibrosis
- Subclinical kidney injury induced by repeated cisplatin administration results in progressive chronic kidney disease
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- C57BL/6 mice require a higher dose of cisplatin to induce renal fibrosis and CCL2 correlates with cisplatin-induced kidney injury
- Suramin protects from cisplatin-induced acute kidney injury
Showing 5 of 7 shared publications
Shenyang Li
Central Arkansas Veterans Healthcare System (US)
6 shared publications
- Alterations of PPARα and its coactivator PGC-1 in cisplatin-induced acute renal failure
- Transgenic expression of proximal tubule peroxisome proliferator–activated receptor-α in mice confers protection during acute kidney injury
- PPAR-α ligand ameliorates acute renal failure by reducing cisplatin-induced increased expression of renal endonuclease G
- Proximal tubule PPARα attenuates renal fibrosis and inflammation caused by unilateral ureteral obstruction
- Increased expression of p21WAF1/CIP1 in kidney proximal tubules mediates fibrosis
Showing 5 of 6 shared publications
Tess V. Dupre
University of Louisville (US)
6 shared publications
- Repeated administration of low-dose cisplatin in mice induces fibrosis
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- Proximal Tubule β2-Adrenergic Receptor Mediates Formoterol-Induced Recovery of Mitochondrial and Renal Function after Ischemia-Reperfusion Injury
- Suramin protects from cisplatin-induced acute kidney injury
- Neutral ceramidase deficiency protects against cisplatin-induced acute kidney injury
Showing 5 of 6 shared publications
István Arany
University of Arkansas for Medical Sciences (US)
5 shared publications
- Activation of ERK or inhibition of JNK ameliorates H2O2 cytotoxicity in mouse renal proximal tubule cells
- STAT3 attenuates EGFR-mediated ERK activation and cell survival during oxidant stress in mouse proximal tubular cells
- CREB mediates ERK-induced survival of mouse renal tubular cells after oxidant stress
- Differentiation-dependent expression of signal transducers and activators of transcription (STATs) might modify responses to growth factors in the cancers of the head and neck
- Prognostic factors in Hungarian breast cancer patients.
Rawad F. Hodeify
Weill Cornell Medical College in Qatar (QA)
5 shared publications
- Gender differences control the susceptibility to ER stress-induced acute kidney injury
- Increased expression of p21WAF1/CIP1 in kidney proximal tubules mediates fibrosis
- Cdk2-dependent phosphorylation of p21 regulates the role of Cdk2 in cisplatin cytotoxicity
- Cdk2 phosphorylation of Bcl-xL after stress converts it to a pro-apoptotic protein mimicking Bax/Bak
- Protection of cisplatin cytotoxicity by an inactive cyclin-dependent kinase
Rick G. Schnellmann
Medical University of South Carolina (US)
5 shared publications
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- 5-HT1F receptor regulates mitochondrial homeostasis and its loss potentiates acute kidney injury and impairs renal recovery
- Suramin protects from cisplatin-induced acute kidney injury
- Suramin: A Potential Therapy for Diabetic Nephropathy
- Suramin Protects From Cisplatin‐Induced Acute Kidney Injury
Parag P. Shah
University of Louisville (US)
5 shared publications
- Repeated administration of low-dose cisplatin in mice induces fibrosis
- Inhibiting glucosylceramide synthase exacerbates cisplatin-induced acute kidney injury
- Suramin protects from cisplatin-induced acute kidney injury
- Neutral ceramidase deficiency protects against cisplatin-induced acute kidney injury
- Suramin Protects From Cisplatin‐Induced Acute Kidney Injury
Sudhir V. Shah
Central Arkansas Veterans Healthcare System
4 shared publications
In database
- PPAR-α ligand ameliorates acute renal failure by reducing cisplatin-induced increased expression of renal endonuclease G
- Cloning and expression of rat caspase-6 and its localization in renal ischemia/reperfusion injury
- Evidence Suggesting a Role of Iron in a Mouse Model of Nephrogenic Systemic Fibrosis
- Iron Chelation Prevents Age‐Related Skeletal Muscle Sarcopenia in Klotho Gene Mutant Mice, a Genetic Model of Aging
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