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Biography and Research Information
OverviewAI-generated summary
A. Edwards' research focuses on molecular mechanisms related to cellular processes and disease models. Current work includes investigating G-quadruplex DNA's effect on the DEAD-box helicase Ded1p's unwinding activity and phase separation, as detailed in a 2021 publication. Additional research efforts are directed toward developing and utilizing animal models for studying respiratory conditions. This includes work on a novel murine partial tracheal occlusion model to investigate altered respiratory dynamics, and targeting CELA1 to improve septation in a mouse hyperoxia model of impaired alveolar development, with publications in 2024 and 2025. Edwards has published 16 papers with 121 citations and an h-index of 3. Key collaborators include Brian M. Varisco and Kevin D. Raney from the University of Arkansas for Medical Sciences, and John C. Marecki from the University of Arkansas at Fayetteville.
Metrics
- h-index: 3
- Publications: 16
- Citations: 124
Selected Publications
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Novel murine partial tracheal occlusion model with altered respiratory dynamics (2025)
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Targeting CELA1 to Improve Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development (2025)
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Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development (2024)
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G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p (2021)
Collaboration Network
Top Collaborators
- Targeting CELA1 to Improve Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Novel murine partial tracheal occlusion model with altered respiratory dynamics
- Targeting CELA1 to Improve Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Novel murine partial tracheal occlusion model with altered respiratory dynamics
- Targeting CELA1 to Improve Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Targeting CELA1 to Improve Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Targeting CELA1 to Improve Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- G-quadruplex DNA inhibits unwinding activity but promotes liquid–liquid phase separation by the DEAD-box helicase Ded1p
- Inhibition of CELA1 Improves Septation in the Mouse Hyperoxia Model of Impaired Alveolar Development