A. M. Jamieson Source Confirmed

Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.

High Impact

Researcher

John Brown University

faculty

46 h-index 218 pubs 10,983 cited

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Biography and Research Information

OverviewAI-generated summary

A faculty member at John Brown University, A. M. Jamieson investigates immune cell function and interactions within the context of pulmonary health and disease. Their research encompasses both experimental and computational approaches, as evidenced by recent work using machine learning to analyze near-infrared spectral fingerprints for macrophage phenotyping. Jamieson's publications also reflect an interest in the role of lung epithelial cell transcriptional regulation in COVID-19-associated coagulopathies, as well as the broader implications of the lung microbiome in cancer and immunity. Additionally, Jamieson's work evaluates prognostic risk models for postoperative pulmonary complications. Their research interests extend to polymer crystallization and properties, surfactants and colloidal systems, and material dynamics, suggesting a multidisciplinary approach to understanding complex biological systems.

Metrics

  • h-index: 46
  • Publications: 218
  • Citations: 10,983

Selected Publications

  • Comparative Analysis of Inflammatory Response in Surgical Wound Drainage Fluid in Scoliosis Surgery: A Study of Neuromuscular vs. Idiopathic Patients (2025) DOI
  • Peripheral Ca <sub>V</sub> 2.2 Channels in the Skin Regulate Prolonged Heat Hypersensitivity during Neuroinflammation (2024) DOI
  • Aspartic proteases are abundant and active in acidified wound fluid (2024) DOI
  • Interleukin-1α links peripheral Ca <sub>V</sub> 2.2 channel activation to rapid adaptive increases in heat sensitivity in skin (2023) DOI
  • IL-1 signaling directs the prioritization of inflammatory sites in a model of post-operative pulmonary infection (2023) DOI
  • IAV/bacteria coinfection causes changes in pathology in an in vitro model of the lung epithelium (2023) DOI
  • Comment on ‘SARS-CoV-2 suppresses anticoagulant and fibrinolytic gene expression in the lung’ (2022) DOI
  • Pulmonary infection interrupts acute cutaneous wound healing through disruption of chemokine signals (2021) DOI
  • Natural killer cells as key regulators of cutaneous wound healing (2021) DOI

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