B
Benjamin Swanson Data-verified
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
High Impact
Researcher
faculty
31 h-index
115 pubs
6,048 cited
Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
Benjamin Swanson's research focuses on cancer biology, particularly pancreatic and colorectal cancers, and the application of advanced techniques to understand these diseases. He investigates mechanisms of drug resistance, such as the role of MUC4 in gemcitabine resistance in pancreatic cancer cells. His work also explores the tumor microenvironment and immune cell populations within tumors, utilizing single-cell RNA sequencing to identify novel cell subsets, like the IDO-1+ tolerogenic dendritic cells found in pancreatic cancer patients but absent in healthy individuals. Additionally, Swanson employs machine learning to analyze complex biological data, such as identifying distinct tumor and stromal cell populations in primary pancreatic tumors through highly-multiplexed immunofluorescence.
His scholarly contributions are evidenced by a substantial body of work, including 115 publications and over 6,000 citations, resulting in an h-index of 31. He is recognized as a highly cited researcher. Swanson collaborates with researchers across institutions, including Hannah C. Hamrick, Charles M. Quick, Michael Cho, and Morgan Middlebrooks. His current research is active, with recent publications extending into 2024 and beyond.
Metrics
- h-index: 31
- Publications: 115
- Citations: 6,048
Selected Publications
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Indirect effects of threat processing on worry: The roles of emotion dysregulation and contrast avoidance (2026)
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The Temporal Dynamics of Attention to Threat and <scp>GAD</scp> Symptoms: A Study of <scp>LPP</scp> Slopes (2025)
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Anxiety Sensitivity and Intolerance of Uncertainty Uniquely Explain the Association of the Late Positive Potential With Generalized Anxiety Disorder Symptoms (2025)
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Explaining Brain-Behavior Relations: Inhibitory Control as an Intermediate Phenotype Between the N2 ERP and the Externalizing Spectrum in Childhood (2024)
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A Developmentally Informed Systematic Review and Meta-Analysis of the Strength of General Psychopathology in Childhood and Adolescence (2023)
Collaboration Network
Top Collaborators
Michael A. Hollingsworth
University of Nebraska Medical Center (US)
14 shared publications
- MUC4 enhances gemcitabine resistance and malignant behaviour in pancreatic cancer cells expressing cancer-associated short O-glycans
- Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors1
- Single-cell RNA-sequencing of human spleens reveals an IDO-1+ tolerogenic dendritic cell subset in pancreatic cancer patients that is absent in normal individuals
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- Humanized Anti-MUC16 Antibody-Conjugated Contrast Agents for Magnetic Resonance Imaging of Pancreatic Cancer
Showing 5 of 14 shared publications
Paul M. Grandgenett
University of Nebraska Medical Center (US)
9 shared publications
- Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors1
- Single-cell RNA-sequencing of human spleens reveals an IDO-1+ tolerogenic dendritic cell subset in pancreatic cancer patients that is absent in normal individuals
- Erratum to: Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
Showing 5 of 9 shared publications
Thomas C. Caffrey
University of Nebraska Medical Center (US)
8 shared publications
- MUC4 enhances gemcitabine resistance and malignant behaviour in pancreatic cancer cells expressing cancer-associated short O-glycans
- Humanized Anti-MUC16 Antibody-Conjugated Contrast Agents for Magnetic Resonance Imaging of Pancreatic Cancer
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
Showing 5 of 8 shared publications
Whitney Goldner
University of Colorado Anschutz Medical Campus (US)
7 shared publications
- Relaxin-2 is a novel biomarker for differentiated thyroid carcinoma in humans
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- Investigating Immune Profiles in Differentiated Thyroid Cancer by Multiplex Immunofluorescence
- Should BRAFV600E be Incorporated into Treatment Recommendations for Thyroid Cancer
- RF11 | PSAT272 Suppressive Tumor Immune Microenvironment is Associated with Aggressive Differentiated Thyroid Cancer
Showing 5 of 7 shared publications
Oleg Shats
University of Nebraska Medical Center (US)
6 shared publications
- Relaxin-2 is a novel biomarker for differentiated thyroid carcinoma in humans
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- Investigating Immune Profiles in Differentiated Thyroid Cancer by Multiplex Immunofluorescence
- Should BRAFV600E be Incorporated into Treatment Recommendations for Thyroid Cancer
- SAT524 Relaxin: A Novel Marker for Pro-Tumoral Thyroid Cancer Microenvironment?
Showing 5 of 6 shared publications
Kelsey Klute
University of Nebraska Medical Center (US)
6 shared publications
- Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors1
- Single-cell RNA-sequencing of human spleens reveals an IDO-1+ tolerogenic dendritic cell subset in pancreatic cancer patients that is absent in normal individuals
- Erratum to: Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors
- Su1675: ROLE OF NUMBER AND SIZE OF NERVES INVOLVED BY PANCREATIC ADENOCARCINOMA IN PREDICTING RECURRENCE AND SURVIVAL
- AI-powered Deep Visual Proteomics reveals critical molecular transitions in pancreatic cancer precursors
Showing 5 of 6 shared publications
Anupam Kotwal
University of Nebraska Medical Center (US)
6 shared publications
- Recurrent Cushing Syndrome From Metastatic Adrenocortical Carcinoma With Fumarate Hydratase Allelic Variant
- Relaxin-2 is a novel biomarker for differentiated thyroid carcinoma in humans
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- RF11 | PSAT272 Suppressive Tumor Immune Microenvironment is Associated with Aggressive Differentiated Thyroid Cancer
- SAT524 Relaxin: A Novel Marker for Pro-Tumoral Thyroid Cancer Microenvironment?
Showing 5 of 6 shared publications
Ana Yuil‐Valdes
University of Nebraska Medical Center (US)
5 shared publications
- Relaxin-2 is a novel biomarker for differentiated thyroid carcinoma in humans
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- Investigating Immune Profiles in Differentiated Thyroid Cancer by Multiplex Immunofluorescence
- RF11 | PSAT272 Suppressive Tumor Immune Microenvironment is Associated with Aggressive Differentiated Thyroid Cancer
- SAT524 Relaxin: A Novel Marker for Pro-Tumoral Thyroid Cancer Microenvironment?
Ernesto Martinez Duarte
University of Florida (US)
4 shared publications
- Recurrent Cushing Syndrome From Metastatic Adrenocortical Carcinoma With Fumarate Hydratase Allelic Variant
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- Investigating Immune Profiles in Differentiated Thyroid Cancer by Multiplex Immunofluorescence
- RF11 | PSAT272 Suppressive Tumor Immune Microenvironment is Associated with Aggressive Differentiated Thyroid Cancer
Krysten E. Vance
University of Nebraska Medical Center (US)
4 shared publications
- Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors1
- Characterization of Immune Infiltrate Along the Leading Edge of Differentiated Thyroid Cancer
- Erratum to: Machine learning analyses of highly-multiplexed immunofluorescence identifies distinct tumor and stromal cell populations in primary pancreatic tumors
- RF11 | PSAT272 Suppressive Tumor Immune Microenvironment is Associated with Aggressive Differentiated Thyroid Cancer
John P. Eggers
4 shared publications
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
Eric C. Collisson
4 shared publications
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
Michelle E. Lewallen
4 shared publications
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
Jarrod R. Tremayne
4 shared publications
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
Judy M. Andersen
4 shared publications
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Data from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
- Supplementary Figure 1 from Cyclin-Dependent Kinase 5 Is Amplified and Overexpressed in Pancreatic Cancer and Activated by Mutant K-Ras
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