Biography and Research Information

OverviewAI-generated summary

Ying Zhong's research centers on investigating molecular mechanisms underlying disease progression and exploring potential therapeutic interventions. Recent publications include work on the role of METTL3 in cell cycle regulation through m⁰A/YTHDF1-dependent pathways, and the inhibitory effects of Ampelopsin on breast cancer cell growth via mitochondrial apoptosis. Zhong has also examined the impact of long-term exposure to PM2.5 on the risk of type 2 diabetes and arthritis in a Chinese cohort, and explored insights into sepsis-induced myocardial dysfunction through bioinformatics analysis of RNA-seq data in mice.

Further research includes the development of multifunctional hydrogels for skin wound healing, utilizing metal-polyphenol capsules. Zhong's work also addresses drug resistance, such as the promotion of cisplatin resistance in nasopharyngeal carcinoma by IAP-1 through inhibition of caspase-3-mediated apoptosis. Additionally, studies have investigated the alleviation of microcirculation disturbance and impaired bone formation by Tanshinol in a rat model, and the creation of surface charge transition nano-theranostics for enhanced photodynamic and photothermal therapy against nasopharyngeal carcinoma.

Zhong leads a research group and has a scholarly profile marked by 138 total publications and 2,062 total citations, with an h-index of 25. The researcher is recognized as highly cited. Key collaborators at the University of Arkansas for Medical Sciences include Umesh D. Wankhade, Henry A. Paz, Craig Porter, and D Sadler, with whom Zhong has co-authored multiple publications.

Metrics

  • h-index: 25
  • Publications: 138
  • Citations: 2,062

Selected Publications

  • Shared and distinct adaptations to early‐life exercise training based on inborn fitness (2025) DOI
  • Maternal high-fat diet-induced obesity in offspring: Unraveling adipose tissue dysfunction mediated by increased heat shock proteins (2025) DOI
  • Shared and distinct adaptations to early-life exercise training based on inborn fitness (2024) DOI
  • Maternal High-Fat Diet-Induced Obesity in Offspring: Unraveling Adipose Tissue Dysfunction Mediated by Increased Heat Shock Proteins (2024) DOI

Collaborators

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